Khursheed Raza

TL;DR: Clinicians and researchers would help clinicians and researchers to understand the tissue pathology, which can help in better planning of the management and avoiding future risks.

TL;DR: It is hypothesize that SARS-CoV-2 host cell entry receptor—ACE2 based mechanism in GI tissue may be involved in COVID-19 in the pathogenesis of digestive symptoms, in increased diabetic complications, and in disease recurrence.

TL;DR: Conclusive remarks of the article make a strong case for plausible role of NGFs in comprehensive regulation of the neurocognitive functions and pathogenesis of related disorders and advocate that future research should be directed to explore use of N GF-based mechanisms in the prevention of implicated diseases as well as to target these molecules pharmacologically.

TL;DR: A review of the progress made since the commencement of the COVID-19 pandemic can be found in this article, where the authors narrate the progress in the human body, including virus-host interactions, pulmonary and other systemic manifestations, immunological dysregulations, complications, host-specific vulnerability, and long-term health consequences in the survivors.

TL;DR: It is proposed that a SARS-CoV-2 binding with ACE2 mediates dysregulation of the sodium dependent nutrient transporters and hence may be a plausible basis for the digestive symptoms in COVID-19 patients.