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Lars M. Blank

Researcher at RWTH Aachen University

Publications -  355
Citations -  10606

Lars M. Blank is an academic researcher from RWTH Aachen University. The author has contributed to research in topics: Chemistry & Pseudomonas putida. The author has an hindex of 49, co-authored 301 publications receiving 8011 citations. Previous affiliations of Lars M. Blank include University of Marburg & Technical University of Dortmund.

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Growth independent rhamnolipid production from glucose using the non-pathogenic Pseudomonas putida KT2440

TL;DR: The metabolic engineering of a rhamnolipid producing Pseudomonas putida KT2440, a strain certified as safety strain using glucose as carbon source to avoid cumbersome product purification, is reported, finding a functional alternative to the pathogen P. aeruginosa.
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Possibilities and limitations of biotechnological plastic degradation and recycling

TL;DR: This Comment aims to clarify important aspects related to myths and realities about plastic biodegradation and suggests distinct strategies for a bio-based circular plastic economy in the future.
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Chemical and biological single cell analysis.

TL;DR: Chemical and biological single cell analyses provide an unprecedented access to the understanding of cell-to-cell differences and basic biological concepts.
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Involvement of Pex13p in Pex14p Localization and Peroxisomal Targeting Signal 2–dependent Protein Import into Peroxisomes

TL;DR: Pex7p and Pex13p functionally interact during PTS2-dependent protein import into peroxisomes, and genetic evidence for the interaction is provided by the observation that overexpression of Px13p suppresses a loss of function mutant of PEx7p.
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TCA cycle activity in Saccharomyces cerevisiae is a function of the environmentally determined specific growth and glucose uptake rates.

Lars M. Blank, +1 more
- 01 Apr 2004 - 
TL;DR: The results indicate that glucose repression of the TCA cycle is regulated by the rates of growth or glucose uptake, or signals derived from these, and while sensing of extracellular glucose concentrations has a general influence on the in vivo TCA Cycle activity, the growth-rate-dependent increase in respiratory T CA cycle activity was independent of glucose sensing.