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Lauren A. Weiss

Researcher at University of California, San Francisco

Publications -  78
Citations -  15034

Lauren A. Weiss is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Autism & Genome-wide association study. The author has an hindex of 39, co-authored 74 publications receiving 11899 citations. Previous affiliations of Lauren A. Weiss include University of California, Berkeley & University of Michigan.

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Variation in ITGB3 has sex-specific associations with plasma lipoprotein(a) and whole blood serotonin levels in a population-based sample.

TL;DR: Variation in ITGB3 in addition to Leu33Pro could contribute to susceptibility to CVD and serotonin in a sex-specific manner, and the results suggest that variation in IT GB3 and its relationship to intermediate phenotypes associated with CVD in the same population is suggested.
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Sex Differences in the Genetic Basis of Morning Serum Cortisol Levels: Genome-Wide Screen Identifies Two Novel Loci Specific to Women

TL;DR: The results suggest that the genetic determinants of morning serum cortisol levels may be different for men and women and that loci on 11p and 14q influence morning cortisol levels in women.
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Integrin β3 genotype influences asthma and allergy phenotypes in the first 6 years of life

TL;DR: The study revealed significant associations between SNPs in ITGB3 and asthma, wheezing, and IgE levels, suggesting an early role for this gene in the development of asthma and allergy.
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Human iPS Cell-Derived Neurons Uncover the Impact of Increased Ras Signaling in Costello Syndrome.

TL;DR: This study is the first to reveal human neuronal pathogenesis resulting from abnormal Ras signaling and provides insights into how these phenotypic abnormalities likely contribute to neurodevelopmental disorders.
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Polymorphisms of the Scavenger Receptor Class B Member 1 Are Associated with Insulin Resistance with Evidence of Gene by Sex Interaction

TL;DR: The data support an association between SCARB1 variants and insulin resistance, especially in women, with evidence of significant gene by sex interaction, and warrant further investigation in additional populations and prompt exploration of a role for SR-BI in the development of insulin resistance.