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Mara J. Dinsmoor

Researcher at NorthShore University HealthSystem

Publications -  57
Citations -  2006

Mara J. Dinsmoor is an academic researcher from NorthShore University HealthSystem. The author has contributed to research in topics: Pregnancy & Gestational age. The author has an hindex of 21, co-authored 51 publications receiving 1744 citations. Previous affiliations of Mara J. Dinsmoor include Case Western Reserve University & Northwestern University.

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Preterm neonatal morbidity and mortality by gestational age: A contemporary cohort

Tracy A. Manuck, +147 more
TL;DR: The data show that there is a continuum of outcomes, with each additional week of gestation conferring survival benefit while reducing the length of initial hospitalization, and these contemporary data can be useful for patient counseling regarding preterm outcomes.
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Risk Factors and Opportunities for Prevention of Early-onset Neonatal Sepsis: A Multicenter Case-Control Study

TL;DR: Either prenatal GBS screening or a risk-based strategy could potentially prevent a substantial portion of GBS cases, and the severity of ampicillin-resistant E colisepsis and its occurrence after maternal antibiotics suggest caution regarding use ofAmpicillin instead of penicillin for GBS prophylaxis.
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A randomized trial of intrapartum versus immediate postpartum treatment of women with intra-amniotic infection

TL;DR: Intrapartum treatment of women with intra‐amniotic infection with ampicillin and gentamicin during labor and immediately after umbilical cord clamping led to a lower incidence of neonatal sepsis and a shorter neonatal hospital stay when compared with postpartum treatment.
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Risk Factors and Opportunities for Prevention of Early-Onset Neonatal Sepsis: A Multicenter Case-Control Study

TL;DR: Either prenatal GBS screening or a risk-based strategy could potentially prevent a substantial portion of GBS cases, but the severity of ampicillin-resistant E coli sepsis and its occurrence after maternal antibiotics suggest caution regarding use ofAmpicillin instead of penicillin for GBS prophylaxis.
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Increased circulating lipid peroxides in severe preeclampsia activate NF-kappaB and upregulate ICAM-1 in vascular endothelial cells.

TL;DR: Elevated plasma levels of lipid peroxides in preeclampsia are associated with increased NF‐κΒ activation and ICAM‐1 expression on HUVEC, which can be inhibited by vitamin E and N‐acetyl‐cysteine.