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Marco Aurélio Romano-Silva

Researcher at Universidade Federal de Minas Gerais

Publications -  261
Citations -  5591

Marco Aurélio Romano-Silva is an academic researcher from Universidade Federal de Minas Gerais. The author has contributed to research in topics: Voltage-dependent calcium channel & Single-nucleotide polymorphism. The author has an hindex of 35, co-authored 247 publications receiving 4909 citations. Previous affiliations of Marco Aurélio Romano-Silva include National Institute of Standards and Technology & Faculdade de Medicina da Universidade Federal de Minas Gerais.

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Decreased in situ expression of interleukin-10 receptor is correlated with the exacerbated inflammatory and cytotoxic responses observed in mucosal leishmaniasis.

TL;DR: The data identified distinct cytokine and cell population profiles for CL versus ML patients and provide a possible mechanism for the development of ML disease through the demonstration that low expression of IL-10 receptor is present in conjunction with a cytotoxic and inflammatory profile in ML.
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Increased serum levels of brain-derived neurotrophic factor in chronic institutionalized patients with schizophrenia

TL;DR: Serum BNDF levels were significantly increased in schizophrenic patients when compared to control subjects, and Interestingly, serum BDNF correlated positively with the clinical scores at the negative subscale of the positive and negative syndrome scale (PANSS).
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Dopamine D2 Receptor Activity Modulates Akt Signaling and Alters GABAergic Neuron Development and Motor Behavior in Zebrafish Larvae

TL;DR: A zebrafish model is used to assess the direct impact of altered dopamine signaling on brain development and larval motor behavior and suggests that D2 receptor signaling suppresses Akt-GSK3b activity, which regulates GABAergic neuron development and motor behavior.
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Increased serum levels of CCL11/eotaxin in schizophrenia.

TL;DR: Higher serum levels of CCL11 in schizophrenia reinforce the view that this disease may be associated with a Th1/Th2 imbalance with a shift toward a Th2 immune response.