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Marco Salvetti

Researcher at Sapienza University of Rome

Publications -  294
Citations -  13327

Marco Salvetti is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Multiple sclerosis & Medicine. The author has an hindex of 44, co-authored 263 publications receiving 10931 citations. Previous affiliations of Marco Salvetti include Max Planck Society & Istituto Neurologico Mediterraneo.

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The tolerability, safety and efficacy of safinamide in elderly Parkinson's disease patients: a retrospective study.

TL;DR: Evidence is provided of the efficacy, tolerability and safety of SF in elderly PD patients and there were no significant differences related to age or daily drug dose in tolerability, safety or efficacy.
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Nerve ultrasonography findings as possible pitfall in differential diagnosis between hereditary transthyretin amyloidosis with polyneuropathy and chronic inflammatory demyelinating polyneuropathy.

TL;DR: It is shown that ATTRv-PN could present the same morphological nerve alterations pattern of CIDP at ultrasonography, adding HRUS findings as a further source of misdiagnosis late-onset ATTRV-PN.
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Antiviral immune response in patients with multiple sclerosis, healthy siblings and twins.

TL;DR: The association between MS status and EBV serology that emerged in some of the tests that have performed should not be overlooked, also considering the lack of substantial genetic, epigenetic and transcriptome variations in identical twins discordant for MS.
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Drugs used in the treatment of multiple sclerosis during COVID-19 pandemic: a critical viewpoint.

TL;DR: In this paper, the authors discuss the disease-modifying agents approved for the treatment of multiple sclerosis (MS) within this context and suggest that the use of these agents may not interfere with the development of COVID-19 and may be considered safe.
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T cell response to N-formylated peptides in humans.

TL;DR: The first evidence of a T lymphocyte response to N‐formylated peptides in humans is presented, which suggests that their expansion in the healthy immune repertoire is independent of the genetic background.