M
Masatoshi Tateno
Researcher at University of California, San Francisco
Publications - 22
Citations - 3366
Masatoshi Tateno is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Virus & Antibody. The author has an hindex of 12, co-authored 21 publications receiving 3314 citations. Previous affiliations of Masatoshi Tateno include Juntendo University.
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Journal ArticleDOI
Vascular Endothelial Growth Factor and Angiopoietin-1 Stimulate Postnatal Hematopoiesis by Recruitment of Vasculogenic and Hematopoietic Stem Cells
Koichi Hattori,Sergio Dias,Beate Heissig,Neil R. Hackett,David Lyden,Masatoshi Tateno,Daniel J. Hicklin,Zhenping Zhu,Larry Witte,Ronald G. Crystal,Malcolm A.S. Moore,Shahin Rafii +11 more
TL;DR: In this paper, VEGF and angiopoietin-1 (Ang-1) levels were elevated by injecting recombinant protein or adenoviral vectors expressing soluble VEGFs165, matrix-bound VEGf189, or Ang-1 into mice, which was associated with an induction of hematopoiesis and increased marrow cellularity.
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Biologic features of HIV-1 that correlate with virulence in the host
TL;DR: The development of disease was found to be correlated with the emergence of HIV-1 variants that were more cytopathic in vitro as the disease progressed and that replicated more efficiently in a wide variety of different human cells.
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Plasma elevation of stromal cell-derived factor-1 induces mobilization of mature and immature hematopoietic progenitor and stem cells.
Koichi Hattori,Beate Heissig,Kei Tashiro,Tasuku Honjo,Masatoshi Tateno,Jae Hung Shieh,Neil R. Hackett,Mannix S. Quitoriano,Ronald G. Crystal,Shahin Rafii,Malcolm A.S. Moore +10 more
TL;DR: It is demonstrated that overexpression of SDF1 in the peripheral circulation results in the mobilization of hematopoietic cells with repopulating capacity, progenitor cells, and precursor cells.
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The Fc and not CD4 receptor mediates antibody enhancement of HIV infection in human cells.
TL;DR: The results indicate that the FcRIII receptor on human macrophages and possibly another Fc receptors on human CD4+ lymphocytes mediate antibody-dependent enhancement of HIV infectivity and that this phenomenon proceeds through a mechanism independent of the CD4 protein.
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Inhibition of both paracrine and autocrine VEGF/ VEGFR-2 signaling pathways is essential to induce long-term remission of xenotransplanted human leukemias
Sergio Dias,Koichi Hattori,Beate Heissig,Zhenping Zhu,Yan Wu,Larry Witte,Daniel J. Hicklin,Masatoshi Tateno,Peter Bohlen,Malcolm A.S. Moore,Shahin Rafii +10 more
TL;DR: Effective antiangiogenic therapies to treat VEGF-producing, VEGFR-expressing leukemias may require blocking both paracrine and autocrine VEGf/VEGFR-2 angiogenic loops to achieve remission and long-term cure.