scispace - formally typeset
M

Masayuki Yoshida

Researcher at Tokyo Medical and Dental University

Publications -  274
Citations -  8554

Masayuki Yoshida is an academic researcher from Tokyo Medical and Dental University. The author has contributed to research in topics: Breast cancer & Medicine. The author has an hindex of 44, co-authored 238 publications receiving 7470 citations. Previous affiliations of Masayuki Yoshida include Kyushu University & Brigham and Women's Hospital.

Papers
More filters
Journal ArticleDOI

MCP-1 and IL-8 trigger firm adhesion of monocytes to vascular endothelium under flow conditions.

TL;DR: It is concluded that chemokines are important modulators of monocyte–endothelial interactions under flow conditions and the finding that IL-8 is a powerful trigger for firm adhesion of monocytes to vascular endothelium reveals an unexpected role for this chemokine in monocyte recruitment.
Journal ArticleDOI

Bone marrow monocyte lineage cells adhere on injured endothelium in a monocyte chemoattractant protein-1-dependent manner and accelerate reendothelialization as endothelial progenitor cells

TL;DR: BM-MLCs can function as EC progenitors that are more potent than CD34+ cells and acquire the ability to adhere on injured endothelium in a MCP-1–dependent manner, leading to reendothelialization associated with inhibition of intimal hyperplasia.
Journal ArticleDOI

Basophils Play a Pivotal Role in Immunoglobulin-G-Mediated but Not Immunoglobulin-E-Mediated Systemic Anaphylaxis

TL;DR: It is shown that basophils, the least common blood cells, were dispensable for IgE-mediated anaphylaxis but played a critical role in IgG-mediated, passive and active systemic anaphYLaxis in mice, and two major, distinct pathways leading to allergen-induced systemic anphylaxis are contrasted.
Journal ArticleDOI

Leukocyte adhesion to vascular endothelium induces E-selectin linkage to the actin cytoskeleton.

TL;DR: Leukocyte adhesion to cytokine-activated HUVEC induces transmembrane cytoskeletal linkage of E-selectin through its cytoplasmic domain, a process which may have important implications for cell-cell signaling as well as mechanical anchoring during leukocyte- endothelial adhesive interactions.
Journal ArticleDOI

HMG-CoA Reductase Inhibitor Modulates Monocyte–Endothelial Cell Interaction Under Physiological Flow Conditions In Vitro: Involvement of Rho GTPase–Dependent Mechanism

TL;DR: An in vitro model that mimicked physiological flow conditions and revealed downregulation of U937 surface expression of CD11a, CD18, and VLA4 after statin treatment has important implications for the lipid-independent effects of statins.