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Matthew C. Tate

Researcher at Northwestern University

Publications -  77
Citations -  3026

Matthew C. Tate is an academic researcher from Northwestern University. The author has contributed to research in topics: Medicine & Speech production. The author has an hindex of 24, co-authored 72 publications receiving 2340 citations. Previous affiliations of Matthew C. Tate include University of California, San Francisco & Georgia Institute of Technology.

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Probabilistic map of critical functional regions of the human cerebral cortex: Broca’s area revisited

TL;DR: This work reports the first bilateral probabilistic map for crucial cortical epicentres of human brain functions in the right and left hemispheres, including sensory, motor, and language (speech, articulation, phonology and semantics), which challenge classical theories of brain organization and provide a distributed framework for future studies of neural networks.
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Biocompatibility of methylcellulose-based constructs designed for intracerebral gelation following experimental traumatic brain injury.

TL;DR: The use of methylcellulose as a scaffolding material, whose concentration and solvent were varied to manipulate its physical properties, indicates that MC is well suited as a biocompatible injectable scaffold for the repair of defects in the brain.
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Preoperative multimodal motor mapping: a comparison of magnetoencephalography imaging, navigated transcranial magnetic stimulation, and direct cortical stimulation.

TL;DR: Navigated TMS is an accurate modality for noninvasively generating preoperative motor maps and maps of the motor system generated with TMS correlate well with those generated by both MEG imaging and DCS.
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Biology of angiogenesis and invasion in glioma

TL;DR: A review of the background biology of the blood brain barrier and its pertinence to blood vessel formation and tumor invasion is presented in this article, where the authors focus their attention on the biology of glioma angiogenesis and invasion, and the key mediators of these processes.
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Laminin and fibronectin scaffolds enhance neural stem cell transplantation into the injured brain.

TL;DR: Behaviour analyses indicated that mice receiving neural stem cells within the laminin‐based scaffold performed significantly better than untreated mice on a spatial learning task, supporting the notion that functional recovery correlates positively with donor cell survival.