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Meng Ye

Researcher at University of Bern

Publications -  27
Citations -  1458

Meng Ye is an academic researcher from University of Bern. The author has contributed to research in topics: Jasmonic acid & Plant defense against herbivory. The author has an hindex of 12, co-authored 23 publications receiving 842 citations. Previous affiliations of Meng Ye include Zhejiang University & Institute of Insect Sciences, Zhejiang University.

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Root exudate metabolites drive plant-soil feedbacks on growth and defense by shaping the rhizosphere microbiota

TL;DR: The results reveal a mechanism by which plants determine the composition of rhizosphere microbiota, plant performance and plant-herbivore interactions of the next generation by modifying root-associated microbiota.
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Antagonism between phytohormone signalling underlies the variation in disease susceptibility of tomato plants under elevated CO2

TL;DR: Under elevated CO2, interactions between tomato and pathogens with different infection strategies were compared and modulated SA/JA cross talk contributes to variation in disease susceptibility under elevatedCO2.
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Plant iron acquisition strategy exploited by an insect herbivore

TL;DR: It is shown that the root-feeding larvae use complexes between iron and benzoxazinoid secondary metabolites to identify maize as a host, to forage within the maize root system, and to increase their growth.
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The Rice Transcription Factor WRKY53 Suppresses Herbivore-Induced Defenses by Acting as a Negative Feedback Modulator of Mitogen-Activated Protein Kinase Activity

TL;DR: Results show that OsWRKY53 functions as a negative feedback modulator of MPK3/MPK6 and thereby acts as an early suppressor of induced defenses, which enables rice plants to control the magnitude of their defensive investment during early signaling.
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Molecular Dissection of Early Defense Signaling Underlying Volatile-Mediated Defense Regulation and Herbivore Resistance in Rice

TL;DR: Herbivore-induced plant volatiles increase plant resistance to herbivores by positively regulating early defense signaling components, including a mitogen-activated protein kinase.