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Michael G. Ziegler

Researcher at University of California, San Diego

Publications -  455
Citations -  23786

Michael G. Ziegler is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Blood pressure & Catecholamine. The author has an hindex of 78, co-authored 455 publications receiving 22509 citations. Previous affiliations of Michael G. Ziegler include San Diego State University & University of Massachusetts Medical School.

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Contributions of the sympathetic nervous system, glutathione, body mass and gender to blood pressure increase with normal aging: influence of heredity.

TL;DR: It is concluded that increases in sympathetic activity contribute to aging-induced SBP Elevations, especially in older females, and BMI increases contribute to BP elevations, particularly in younger subjects.
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Relationship between cerebrospinal fluid norepinephrine and blood pressure in neurologic patients.

TL;DR: For instance, in 126 patients hospitalized for various diseases norepinephrine in cerebrospinal fluid correlated with blood pressure (r = 0.41, p < 0.0001) and heart rate as mentioned in this paper.
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Angiotensin II in adrenergic-induced alterations in glomerular hemodynamics

TL;DR: Renal nerve stimulation can regulate glomerular ultrafiltration by altering vascular resistances, and angiotensin II appears to be a critical factor for the full functional expression of renal nerve stimulation at the glomerulus.
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Refining caregiver vulnerability for clinical practice: determinants of self-rated health in spousal dementia caregivers.

TL;DR: Caregivers with poor/fair SRH were characterized by higher levels of medical comorbidity, low physical function, high negative, but low positive affect and longer duration of caregiving, as well as more severe dementia and greater functional impairment of the care recipient.
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Enumerative immune changes following acute stress: effect of the menstrual cycle

TL;DR: The findings suggest that the changes in reproductive hormones associated with the menstrual cycle have no appreciable effect on lymphocyte numbers or their response to acute stress.