M
Michael G. Ziegler
Researcher at University of California, San Diego
Publications - 455
Citations - 23786
Michael G. Ziegler is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Blood pressure & Catecholamine. The author has an hindex of 78, co-authored 455 publications receiving 22509 citations. Previous affiliations of Michael G. Ziegler include San Diego State University & University of Massachusetts Medical School.
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Journal ArticleDOI
Sleep Deprivation Potentiates Activation of Cardiovascular and Catecholamine Responses in Abstinent Alcoholics
TL;DR: Partial night sleep deprivation induces elevated heart rate and sympathetic catecholamine responses in alcoholic subjects as compared with controls, and this sympathetic activation is sustained after nights of partial and recovery sleep.
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Attenuation of T-lymphocyte demargination and adhesion molecule expression in response to moderate exercise in physically fit individuals
Suzi Hong,Todd A. Johnson,Noha H. Farag,H. J. Guy,Scott C. Matthews,Michael G. Ziegler,Paul J. Mills +6 more
TL;DR: It is suggested that physical fitness attenuates demargination of selected lymphocyte subsets in response to moderate exercise, and being physically fit may offset exaggerated immune cell responses to stress.
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Menstrual cycle and ovarian hormone effects on plasma and platelet monoamine oxidase (MAO) and plasma dopamine-beta-hydroxylase (DBH) activities in the rhesus monkey.
TL;DR: The ovariectomized females showed significant differences from the controls, confirming the effects of the ovarian sex steroid hormones on platelet MAO and plasma DBH and variation in these peripheral enzymes may be reflective of changes in brain monoamine systems.
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Endogenous opioid inhibition and facilitation of gonadotropin-releasing hormone release from the median eminence in vitro: potential role of catecholamines.
TL;DR: The precise correlation between GnRH and DA/NE release suggests that the catecholamine terminals close to both GnRH- and endorphin-containing terminals in the ME may mediate this opioid regulation.
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The effect of hypoxia on baroreflexes and pressor sensitivity in sleep apnea and hypertension.
Michael G. Ziegler,Richard A. Nelesen,Paul J. Mills,Sonia Ancoli-Israel,Jack L. Clausen,Lana L. Watkins,Joel E. Dimsdale +6 more
TL;DR: These studies suggest that apneics could have an exaggerated pressor sensitivity to norepinephrine, and might also have difficulty returning BP to normal levels, because hypoxia impaired baroreflexes.