M
Michael G. Ziegler
Researcher at University of California, San Diego
Publications - 455
Citations - 23786
Michael G. Ziegler is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Blood pressure & Catecholamine. The author has an hindex of 78, co-authored 455 publications receiving 22509 citations. Previous affiliations of Michael G. Ziegler include San Diego State University & University of Massachusetts Medical School.
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Journal ArticleDOI
Leptin is related to epinephrine levels but not reproductive hormone levels in cycling African-American and Caucasian women
TL;DR: In women leptin levels are not associated with cyclic changes in estrogen or progesterone but may be associated with catecholamine levels, and independent of BMI, leptin was negatively correlated with plasma epinephrine levels.
Journal Article
Neuropeptide Y-Mediated Pressor Responses Following High-Frequency Stimulation of the Rat Sympathetic Nervous System
TL;DR: The rapid blood pressure spikes that occur during electrical stimulation are attenuated by alpha adrenergic but not by NPY antagonists, and there is a prolonged pressor response after high-frequency stimulation of the sympathetic trunk in pithed rats that begins after 1 to 2 min of stimulation and lasts approximately 10 min after the end of stimulation.
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Ultradian variations of chromogranin A in humans
Marwan A. Takiyyuddin,H. P. H. Neumann,Justine H. Cervenka,Brian P. Kennedy,Thai Dinh,Michael G. Ziegler,A. D. Baron,Daniel T. O'Connor +7 more
TL;DR: Somatostatin, a widespread inhibitor of nonsympathoadrenal neuroendocrine secretion, diminished both the frequency and amplitude of plasma CgA peaks, and spontaneous variations in basal C gA are not directly linked to alterations in sympathoadrennal catecholamine secretion.
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Cerebrospinal fluid norepinephrine alterations during electrical stimulation of cerebellar, cerebral surfaces in epileptic patients
TL;DR: This study suggests that electrical stimulation of the anterodorsal cerebellum in man evokes alterations in noradrenergic metabolism and Cerebellar stimulation-induced elevations in norepinephrine may inhibit cerebellar, cerebral, and spinal neuronaf activity.
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Subjective social status predicts in vivo responsiveness of β-adrenergic receptors.
TL;DR: In this paper, subjective social status (SSS) and β-AR function was examined in the context of the Chronotropic 25 dose (CD25) in 94 healthy participants.