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Nicola Abate

Researcher at University of Texas Medical Branch

Publications -  109
Citations -  8706

Nicola Abate is an academic researcher from University of Texas Medical Branch. The author has contributed to research in topics: Insulin resistance & Adipose tissue. The author has an hindex of 42, co-authored 109 publications receiving 8069 citations. Previous affiliations of Nicola Abate include University of Texas Southwestern Medical Center & Shriners Hospitals for Children - Galveston.

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Metabolic Dysregulation and Adipose Tissue Fibrosis: Role of Collagen VI†

TL;DR: It is suggested that weakening the extracellular scaffold of adipocytes enables their stress-free expansion during states of positive energy balance, which is consequently associated with an improved inflammatory profile and implicating “adipose tissue fibrosis” as a hallmark of metabolically challenged adipocytes.
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Relationships of generalized and regional adiposity to insulin sensitivity in men.

TL;DR: Subcutaneous truncal fat plays a major role in obesity-related insulin resistance in men, whereas intraperitoneal fat and retroperitoneAL fat have a lesser role.
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The metabolic syndrome and uric acid nephrolithiasis: Novel features of renal manifestation of insulin resistance

TL;DR: It is concluded that one renal manifestation of insulin resistance may be low urinary ammonium and pH, which can result in increased risk of uric acid precipitation despite normouricosuria.
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Relationship between generalized and upper body obesity to insulin resistance in Asian Indian men

TL;DR: Results show that Asian Indian men are more insulin resistant than Caucasian men independently of generalized or truncal adiposity, which may account for the excessive morbidity and mortality from diabetes and coronary heart disease in this population.
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Obesity, adiposity, and dyslipidemia: A consensus statement from the National Lipid Association

TL;DR: On September 16, 2012, the National Lipid Association held a Consensus Conference with the goal of better defining the effect of adiposity on lipoproteins, how the pathos of excessive body fat (adiposopathy) contributes to dyslipidemia, and how therapies such as appropriate nutrition, increased physical activity, weight-management drugs, and bariatric surgery might be expected to impact dys Lipidemia.