O
Olivier Bernard
Researcher at French Institute for Research in Computer Science and Automation
Publications - 830
Citations - 42407
Olivier Bernard is an academic researcher from French Institute for Research in Computer Science and Automation. The author has contributed to research in topics: Liver transplantation & Segmentation. The author has an hindex of 96, co-authored 790 publications receiving 37878 citations. Previous affiliations of Olivier Bernard include Intelligence and National Security Alliance & Institut national des sciences appliquées.
Papers
More filters
Journal ArticleDOI
TEL-JAK2 transgenic mice develop T-cell leukemia.
Clémence Carron,Françoise Cormier,Françoise Cormier,Anne Janin,Anne Janin,Virginie Lacronique,Virginie Lacronique,Marco Giovannini,Marco Giovannini,Marie-Thérèse Daniel,Marie-Thérèse Daniel,Olivier Bernard,Olivier Bernard,Jacques Ghysdael,Jacques Ghysdael +14 more
TL;DR: It is concluded that the TEL-JAK2 fusion is an oncogene in vivo and that its expression in lymphoid cells results in the preferential expansion of CD8-positive T cells.
Journal ArticleDOI
Pulmonary arteriovenous shunting in children with liver disease
Thierry Barbé,Jean Losay,Gilles Grimon,Denis Devictor,Anne Sardet,Frédéric Gauthier,Didier Houssin,Olivier Bernard +7 more
TL;DR: It is indicated that systematic screening for PAVS should be part of the examination of children with portal hypertension and that the risk is highest and earliest in children with biliary atresia and polysplenia syndrome and the prognosis may be related to the level of PaO2 while the patient is breathing 100% O2.
Journal ArticleDOI
HOX11L2 expression defines a clinical subtype of pediatric T-ALL associated with poor prognosis.
Paola Ballerini,Annick Blaise,Maryvonne Busson-Le Coniat,Xin Ying Su,Jessica Zucman-Rossi,Mircea Adam,Jacqueline Van Den Akker,Christine Perot,Beatrice Pellegrino,Judith Landman-Parker,Luc Douay,Roland Berger,Olivier Bernard +12 more
TL;DR: The data show that HOX11L2 expression was a suitable marker for minimal residual disease follow-up and was significantly associated with relapse (P =.02), and the pathogenesis of this type of hematologic malignancy was analyzed.
Journal ArticleDOI
RHOA G17V Induces T Follicular Helper Cell Specification and Promotes Lymphomagenesis.
Jose R. Cortes,Alberto Ambesi-Impiombato,Lucile Couronné,Lucile Couronné,S. Aidan Quinn,Christine S Kim,Ana Carolina da Silva Almeida,Zachary West,Laura Belver,Marta Sanchez Martin,Laurianne Scourzic,Laurianne Scourzic,Laurianne Scourzic,Govind Bhagat,Olivier Bernard,Olivier Bernard,Olivier Bernard,Adolfo A. Ferrando,Adolfo A. Ferrando,Teresa Palomero,Teresa Palomero +20 more
TL;DR: Tet2-/-RHOA G17V tumor proliferation in vivo can be inhibited by ICOS/PI3K-specific blockade, supporting a driving role for ICOS signaling in Tfh cell transformation, and expression together with Tet2 loss resulted in development of AITL in mice.
Journal ArticleDOI
Incidence and prognostic value of TET2 alterations in de novo acute myeloid leukemia achieving complete remission.
Olivier Nibourel,Olivier Kosmider,Meyling Cheok,Nicolas Boissel,Aline Renneville,Nathalie Philippe,Hervé Dombret,François Dreyfus,Bruno Quesnel,Sandrine Geffroy,Samuel Quentin,Catherine Roche-Lestienne,Jean-Michel Cayuela,Christophe Roumier,Pierre Fenaux,William Vainchenker,Olivier Bernard,Jean Soulier,Michaela Fontenay,Claude Preudhomme +19 more
TL;DR: TET2 gene status was not significantly correlated with disease-free survival and overall survival, both in the entire cohort and in patients with normal karyotype.