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Patrizia Brigidi

Researcher at University of Bologna

Publications -  273
Citations -  21389

Patrizia Brigidi is an academic researcher from University of Bologna. The author has contributed to research in topics: Gut flora & Microbiome. The author has an hindex of 69, co-authored 243 publications receiving 17599 citations. Previous affiliations of Patrizia Brigidi include University of Catania & Leiden University Medical Center.

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Enteral versus Parenteral Nutrition as Nutritional Support after Allogeneic Hematopoietic Stem Cell Transplantation: a Systematic Review and Meta-Analysis.

TL;DR: In this paper, the authors provide an up-to-date systematic review and meta-analysis of studies comparing EN and PN for nutritional support during the neutropenic period after allogeneic hematopoietic stem cell transplantation (allo-HSCT).
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Transcriptomic clues to understand the growth of Lactobacillus rhamnosus in cheese

TL;DR: A transcriptomic approach was used to identify genes potentially contributing to the growth ability of L. rhamnosus PR1019 in cheese-like medium (CB) using a Transcriptomic approach, based on cDNA-amplified fragment length polymorphism (cDNA-AFLP) and quantitative real-time reverse transcription-PCR (qPCR).
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Novel Targets of Sulforaphane in Primary Cardiomyocytes Identified by Proteomic Analysis

TL;DR: New insights into the anti-glycative role of sulforaphane in cardiomyocytes are given, confirming its pleiotropic behavior in counteracting cardiovascular diseases.
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Simultaneous HS-SPME GC-MS determination of short chain fatty acids, trimethylamine and trimethylamine N-oxide for gut microbiota metabolic profile

TL;DR: The presented method is successfully applied to the quantitation of the considered gut metabolites in faecal samples from Italian healthy volunteers and adequate sensitivity and precision were achieved for all the target analytes.
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A Versatile New Model of Chemically Induced Chronic Colitis Using an Outbred Murine Strain.

TL;DR: The results show that the intrarectal administration of DNBS effectively causes colitis in both female and male CD-1 mice in a dose-dependent manner, as reflected by loss of body mass, macroscopic scores and histological scores, and colon cytokine levels and mesenteric lymph node characteristics indicate that this model involves immune system activation.