P
Philip A. Poole-Wilson
Researcher at National Institutes of Health
Publications - 443
Citations - 69648
Philip A. Poole-Wilson is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Heart failure & Heart disease. The author has an hindex of 105, co-authored 443 publications receiving 66861 citations. Previous affiliations of Philip A. Poole-Wilson include Harefield Hospital & St George's Hospital.
Papers
More filters
Journal ArticleDOI
Regulation of intracellular pH in the myocardium; relevance to pathology.
TL;DR: In man acidosis can be detected 15 s after the occlusion of a coronary artery and is a major mechanism for the simultaneous loss of contractility in ischaemia, and the role of acidosis in causing cell necrosis is not established.
The Task Force for the Diagnosis and Treatment of Acute and Chronic Heart Failure 2008 of the European Society of Cardiology. Developed in collaboration with the Heart Failure Association of the ESC (HFA) and endorsed by the European Society of Intensive Care Medicine (ESICM)
Alain Cohen-Solal,Piotr Ponikowski,Philip A. Poole-Wilson,Anna Strömberg,Dirk J. van Veldhuisen,Arno W. Hoes,Markku S. Nieminen,Karl Swedberg +7 more
TL;DR: The ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure 2008 as mentioned in this paper were developed in collaboration with the Heart Failure Association of the ESC (HFA) and endorsed by the European Society of Intensive Care Medicine (ESICM).
Journal ArticleDOI
The underreporting of results and possible mechanisms of ‘negative’ drug trials in patients with chronic heart failure
TL;DR: Large drug trials have become very important to determine which drugs should be used in the treatment of patients with chronic heart failure (CHF), and these trials that were conducted in the last 10 years are discussed to provide insight into the pathophysiology and treatment options in CHF.
Journal ArticleDOI
Endothelial control of lower limb blood flow in chronic heart failure.
TL;DR: There is reduced blood flow and raised vascular resistance at rest in the legs of patients with heart failure and the degree of impaired blood flow in the leg correlates with the severity of heart failure.
Journal ArticleDOI
Authors' response to “Stress (Takotsubo) cardiomyopathy—a novel pathophysiological hypothesis to explain catecholamine-induced acute myocardial stunning”
TL;DR: Authors' response to “Stress (Takotsubo) cardiomyopathy—a novel pathophysiological hypothesis to explain catecholamine-induced acute myocardial stunning”.