P
Philip A. Poole-Wilson
Researcher at National Institutes of Health
Publications - 443
Citations - 69648
Philip A. Poole-Wilson is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Heart failure & Heart disease. The author has an hindex of 105, co-authored 443 publications receiving 66861 citations. Previous affiliations of Philip A. Poole-Wilson include Harefield Hospital & St George's Hospital.
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Journal ArticleDOI
Response to Letter Regarding Article, “Anabolic Deficiency in Men With Chronic Heart Failure: Prevalence and Detrimental Impact on Survival”
Ewa A. Jankowska,Bartosz Biel,Jacek Majda,Waldemar Banasiak,Piotr Ponikowski,Monika Lopuszanska,Alicja Szklarska,Stefan D. Anker,Philip A. Poole-Wilson,Marek Medras +9 more
TL;DR: In male CHF patients, anabolic hormone depletion is common, and a deficiency of each anabolic hormones is an independent marker of poor prognosis.
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Calcium out of control.
TL;DR: The results suggest that the uptake of calcium on reoxygenation or reperfusion is related to the reintroduction of oxygen and caused by an increased calcium influx down the concentration gradient.
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Haemoglobin predicts survival in patients with chronic heart failure: a substudy of the ELITE II trial
Rakesh Sharma,Darrel P. Francis,Bertram Pitt,Philip A. Poole-Wilson,Andrew J.S. Coats,Stefan D. Anker +5 more
TL;DR: This work aims to investigate the relationship between the concentration of haemoglobin and survival in chronic heart failure and to correct anaemia in patients with CHF.
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Reduced contraction and altered frequency response of isolated ventricular myocytes from patients with heart failure.
TL;DR: Reduced contraction, slowed relaxation, and impaired frequency response occurring at the level of the individual ventricular myocyte can be demonstrated in human heart failure, demonstrating that disruption of myocyte function can contribute to both the systolic and the diastolic abnormalities that occur in the failing human heart.
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Heart rate response during exercise predicts survival in adults with congenital heart disease.
Gerhard-Paul Diller,Konstantinos Dimopoulos,Darlington O. Okonko,Anselm Uebing,Craig S. Broberg,Sonya V. Babu-Narayan,Stephanie Bayne,Philip A. Poole-Wilson,Richard Sutton,Darrel P. Francis,Michael A. Gatzoulis +10 more
TL;DR: An abnormal heart rate response to exercise identifies ACHD patients with a higher risk of mortality in the midterm, even after accounting for antiarrhythmic medication and exercise capacity.