P
Priscila Lupino Gratão
Researcher at Sao Paulo State University
Publications - 99
Citations - 4773
Priscila Lupino Gratão is an academic researcher from Sao Paulo State University. The author has contributed to research in topics: Glutathione reductase & Catalase. The author has an hindex of 31, co-authored 80 publications receiving 3885 citations. Previous affiliations of Priscila Lupino Gratão include University of São Paulo & Escola Superior de Agricultura Luiz de Queiroz.
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Making the life of heavy metal-stressed plants a little easier
TL;DR: This review is to assess the mode of action and role of antioxidants in protecting plants from stress caused by the presence of heavy metals in the environment.
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Phytoremediation: green technology for the clean up of toxic metals in the environment
Priscila Lupino Gratão,Majeti Narasimha Vara Prasad,Patrícia Felippe Cardoso,Peter J. Lea,Ricardo Antunes Azevedo +4 more
TL;DR: In this paper, the use of naturally occurring metal tolerant plants and the application of genetic manipulation, should hasten the process of transferring this technology from laboratory to field, and it is essential to investigate and understand how plants are able to tolerate toxic metals and to identify which metabolic pathways and genes are involved in such a process.
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Acquired tolerance of tomato (Lycopersicon esculentum cv. Micro‐Tom) plants to cadmium‐induced stress
Priscila Lupino Gratão,Carolina C. Monteiro,A.M. Antunes,Lázaro Eustáquio Pereira Peres,Ricardo Antunes Azevedo +4 more
TL;DR: The effects of varying concentrations of cadmium (Cd) on the development of Lycopersicon esculentum cv.
Journal ArticleDOI
Antioxidant metabolism of coffee cell suspension cultures in response to cadmium
Rui A. Gomes-Junior,Carlos Alberto Moldes,Fabricio S. Delite,Georgia Bertoni Pompeu,Priscila Lupino Gratão,Paulo Mazzafera,Peter J. Lea,Ricardo Antunes Azevedo +7 more
TL;DR: The results suggest that the higher concentrations of CdCl(2) may lead to oxidative stress, which may also be related to the inhibition of APX activity probably due to glutathione and ascorbate depletion.