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Ranjan Deka

Researcher at University of Cincinnati

Publications -  197
Citations -  10628

Ranjan Deka is an academic researcher from University of Cincinnati. The author has contributed to research in topics: Population & Allele. The author has an hindex of 52, co-authored 178 publications receiving 9856 citations. Previous affiliations of Ranjan Deka include University of Texas Health Science Center at Houston & Boston Children's Hospital.

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Smoking and family history and risk of aneurysmal subarachnoid hemorrhage.

TL;DR: Evidence of a gene–environment interaction with smoking exists for aneurysmal subarachnoid hemorrhage, important to counseling family members and for screening of intracranial aneurYSm (IA) as well as the design and interpretation of genetic epidemiology of IA studies.
Journal Article

Characteristics of polymorphism at a VNTR locus 3' to the apolipoprotein B gene in five human populations.

TL;DR: Analysis of allele frequency distribution at the hypervariable locus 3' to the apolipoprotein B gene (ApoB 3' VNTR) in five well-defined human populations by using the PCR technique reveals that, in general, the allele frequency distributions at this locus are in agreement with the predictions of the classical mutation-drift models.
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Microsatellite Data Support an Early Population Expansion in Africa

TL;DR: Analysis of three data sets from surveys of microsatellite loci in ethnographically defined populations reveals that most of the African populations analyzed, but none of the 30 non-African populations showed PK distributions with nonzero peaks, which indicate either an earlier expansion or a larger effective population size for African populations.
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Association of Apolipoprotein E4 and Haplotypes of the Apolipoprotein E Gene With Lobar Intracerebral Hemorrhage

TL;DR: Haplotype association analysis demonstrated a significant association of the apo E gene with lobar ICH among whites (P<0.0001) and blacks (P=0.0024), while genotyped 12 single-nucleotide polymorphisms in the 5′ upstream regulatory, exonic, and intronic regions of the APO E gene and performed genotype and haplotype association analyses.
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Genetics and biology of human ovarian teratomas. III. Cytogenetics and origins of malignant ovarian germ cell tumors.

TL;DR: The analysis of centromeric heteromorphisms and DNA fingerprints of host and teratoma using the M13 probe suggests that germ cell teratomas could arise by the fusion of two ova.