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Richard F. Spaide

Researcher at Manhattan Eye, Ear and Throat Hospital

Publications -  444
Citations -  42209

Richard F. Spaide is an academic researcher from Manhattan Eye, Ear and Throat Hospital. The author has contributed to research in topics: Macular degeneration & Fluorescein angiography. The author has an hindex of 98, co-authored 422 publications receiving 36946 citations. Previous affiliations of Richard F. Spaide include University of Illinois at Chicago & Landstuhl Regional Medical Center.

Papers
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Evaluation of Segmentation of the Superficial and Deep Vascular Layers of the Retina by Optical Coherence Tomography Angiography Instruments in Normal Eyes.

TL;DR: Evaluating the segmentation of retinal layers among 3 OCT angiography instruments in the central macula, an area where the superficial and deep vascular plexuses terminate, found none of the instruments produced segmented regions that followed the relevant anatomic layers correctly.
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Type 3 neovascularization: Evolution, association with pigment epithelial detachment, and treatment response as revealed by spectral domain optical coherence tomography

TL;DR: In some eyes with age-related macular degeneration, the earliest sign of Type 3 neovascularization is punctate hyperreflective foci above the external limiting membrane, and the mature Type 3 lesions and associated serous pigment epithelial detachments are highly responsive to anti-vascular endothelial growth factor therapy.
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Fundus autofluorescence and visual acuity in central serous chorioretinopathy.

TL;DR: The FAF abnormalities in CSC show multiple distinct patterns and seem to provide functional information and their correlations with corrected visual acuity are evaluated.
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Comparing functional and morphologic characteristics of lamellar macular holes with and without lamellar hole-associated epiretinal proliferation.

TL;DR: Eyes with LMH and LHEP were associated with poorer visual acuity, larger LMH diameters, thinner retinal thickness, and higher incidence of ellipsoid disruption compared with eyes without LHEP, suggesting a process involving more severe retinal tissue loss and injury.