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Richard J. Davidson

Researcher at University of Wisconsin-Madison

Publications -  642
Citations -  99052

Richard J. Davidson is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Prefrontal cortex & Mindfulness. The author has an hindex of 156, co-authored 602 publications receiving 91414 citations. Previous affiliations of Richard J. Davidson include Iowa State University & French Institute of Health and Medical Research.

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Altered anterior insula activation during anticipation and experience of painful stimuli in expert meditators

TL;DR: It is suggested that cultivating experiential openness down-regulates anticipatory representation of aversive events, and increases the recruitment of attentional resources during pain, which is associated with faster neural habituation.
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How and why do the two cerebral hemispheres interact

TL;DR: The data suggest that when both hemispheres have some competence at a difficult task, there is a benefit to interhemispheric interaction and the role of the CC in the dynamic distribution of attention may be particularly relevant to this advantage.
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Context-specific freezing and associated physiological reactivity as a dysregulated fear response.

TL;DR: The hypothesis that only behaviors under the less threatening context would be associated with higher cortisol and sympathetic cardiac activity was confirmed and only task-specific freezing behavior predicted higher reactive and basal cortisol levels and resting PEP measured 1 week later.
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BOLD signal in insula is differentially related to cardiac function during compassion meditation in experts vs. novices.

TL;DR: Data confirm that compassion enhances the emotional and somatosensory brain representations of others' emotions, and that this effect is modulated by expertise.
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Purpose in life predicts better emotional recovery from negative stimuli.

TL;DR: A proximal mechanism by which purpose in life may afford protection from negative events and confer resilience is through enhanced automatic emotion regulation after negative emotional provocation.