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Rita Vos

Researcher at Katholieke Universiteit Leuven

Publications -  327
Citations -  11029

Rita Vos is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Gastric distension & Distension. The author has an hindex of 54, co-authored 327 publications receiving 10418 citations. Previous affiliations of Rita Vos include University of Oxford & TSMC.

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Physiological consequences of loss of plasminogen activator gene function in mice.

TL;DR: Direct evidence suggests a crucial role for the fibrinolytic system and its physiological triggers, tissue-type and urokinase-type (u-PA) plasminogen activator, in many proteolytic processes.
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Deletion of NEMO/IKKγ in Liver Parenchymal Cells Causes Steatohepatitis and Hepatocellular Carcinoma

TL;DR: NEMO-mediated NF-κB activation in hepatocytes has an essential physiological function to prevent the spontaneous development of steatohepatitis and hepatocellular carcinoma, identifying NEMO as a tumor suppressor in the liver.
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Protection of hepatocyte mitochondrial ultrastructure and function by strict blood glucose control with insulin in critically ill patients.

TL;DR: Strict glycaemic control with intensive insulin therapy prevented or reversed ultrastructural and functional abnormalities of hepatocyte mitochondria and the lack of effect on skeletal-muscle mitochondria suggests a direct effect of glucose toxicity and glucose control, rather than of insulin, as the likely explanation.
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Morphological and biochemical characterization of a human liver in a uPA-SCID mouse chimera.

TL;DR: The repopulation, organization, and function of human hepatocytes in a mouse recipient and the infections with hepatitis B virus (HBV) and hepatitis C virus (HCV) of the transplanted cells are described.
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Clinicopathological study on cholangiolocellular carcinoma suggesting hepatic progenitor cell origin

TL;DR: In this paper, the authors investigated the clinicopathological features of 30 cholangiolocellular carcinoma (CLC) cases and their relationship to hepatic progenitor cells (HPCs) using immunohistochemistry and electron microscopy.