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Robert T. Schultz

Researcher at Children's Hospital of Philadelphia

Publications -  269
Citations -  28251

Robert T. Schultz is an academic researcher from Children's Hospital of Philadelphia. The author has contributed to research in topics: Autism & Autism spectrum disorder. The author has an hindex of 71, co-authored 257 publications receiving 24640 citations. Previous affiliations of Robert T. Schultz include University of Pennsylvania & Northwestern University.

Papers
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Visual fixation patterns during viewing of naturalistic social situations as predictors of social competence in individuals with autism.

TL;DR: A novel method of quantifying atypical strategies of social monitoring in a setting that simulates the demands of daily experience is reported, finding that fixation times on mouths and objects but not on eyes are strong predictors of degree of social competence.
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The Social Motivation Theory of Autism

TL;DR: It is concluded that ASD can be construed as an extreme case of diminished social motivation and, as such, provides a powerful model to understand humans' intrinsic drive to seek acceptance and avoid rejection.
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Autism genome-wide copy number variation reveals ubiquitin and neuronal genes

TL;DR: Several new susceptibility genes encoding neuronal cell-adhesion molecules, including NLGN1 and ASTN2, were enriched with CNVs in ASD cases compared to controls, and duplications 55 kilobases upstream of complementary DNA AK123120 indicate that these two important gene networks expressed within the central nervous system may contribute to the genetic susceptibility of ASD.
Book

Autism and Pervasive Developmental Disorders

TL;DR: The quantity and quality of research into autism and related conditions have increased dramatically in recent years as mentioned in this paper, and there has been significant advances in the molecular genetics of autism and understanding of the underlying neurobiological processes.
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Developmental deficits in social perception in autism: the role of the amygdala and fusiform face area.

TL;DR: It is argued that the development of face perception and social cognitive skills are supported by the amygdala–fusiform system, and that deficits in this network are instrumental in causing autism.