R
Rudolf K. Braun
Researcher at University of Wisconsin-Madison
Publications - 28
Citations - 1464
Rudolf K. Braun is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Hyperoxia & Lung injury. The author has an hindex of 16, co-authored 26 publications receiving 1195 citations. Previous affiliations of Rudolf K. Braun include Loyola University Chicago.
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Journal ArticleDOI
IL-17–dependent cellular immunity to collagen type V predisposes to obliterative bronchiolitis in human lung transplants
William J. Burlingham,Robert B. Love,Robert B. Love,Ewa Jankowska-Gan,Lynn D. Haynes,Qingyong Xu,Joseph L. Bobadilla,Keith C. Meyer,Mary S. Hayney,Rudolf K. Braun,Rudolf K. Braun,Daniel S. Greenspan,Bagavathi Gopalakrishnan,Junchao Cai,David D. Brand,Shigetoshi Yoshida,Oscar W. Cummings,David S. Wilkes +17 more
TL;DR: While alloimmunities initiates lung transplant rejection, de novo autoimmunity mediated by col(V)-specific Th17 cells and monocyte/macrophage accessory cells ultimately causes progressive airway obliteration.
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Direct human health risks of increased atmospheric carbon dioxide
Tyler Jacobson,Jasdeep S. Kler,Michael T. Hernke,Rudolf K. Braun,Keith C. Meyer,William E. Funk +5 more
TL;DR: Preliminary evidence indicates potential health risks at CO2 exposures as low as 1,000 ppm—a threshold that is already exceeded in many indoor environments with increased room occupancy and reduced building ventilation rates, and equivalent to some estimates for urban outdoor air concentrations before 2100.
Journal ArticleDOI
IL-17 Producing γδ T Cells are Required for a Controlled Inflammatory Response after Bleomycin-induced Lung Injury
Rudolf K. Braun,Christina Ferrick,Paul Neubauer,Michael W. Sjoding,Anja Sterner-Kock,Martin Kock,Lei F. Putney,David A. Ferrick,Dallas M. Hyde,Robert B. Love +9 more
TL;DR: Mouse γδ T cells produce IL-17 in response to lung injury and are required for an organized inflammatory response and epithelial repair, and the lack of γ Δ T cells correlates with increased inflammation and fibrosis.
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Th-17, Monokines, Collagen Type V, and Primary Graft Dysfunction in Lung Transplantation
Joseph L. Bobadilla,Robert B. Love,Ewa Jankowska-Gan,Qingyong Xu,Lynn D. Haynes,Rudolf K. Braun,Mary S. Hayney,Alejandro Munoz del Rio,Keith C. Meyer,Daniel S. Greenspan,Jose R. Torrealba,Kathleen M. Heidler,Oscar W. Cummings,Takekazu Iwata,David D. Brand,Robert G. Presson,William J. Burlingham,David S. Wilkes,David S. Wilkes +18 more
TL;DR: The data suggest that activation of innate immunity by col(V)-specific Th-17 memory cells represents a novel pathway to PGD after lung transplantation.
Journal ArticleDOI
Cellular basis of tissue regeneration by omentum.
Shivanee Shah,Erin M. Lowery,Rudolf K. Braun,Alicia Martin,Nick Huang,Melissa Medina,Periannan Sethupathi,Yoichi Seki,Mariko Takami,Kathryn R Byrne,Christopher H. Wigfield,Robert B. Love,Makio Iwashima +12 more
TL;DR: It is proposed that the omentum is a designated organ for tissue repair and healing in response to foreign invasion and tissue damage.