R
Russell M. Lebovitz
Researcher at Baylor College of Medicine
Publications - 34
Citations - 3484
Russell M. Lebovitz is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Gene & RNA. The author has an hindex of 21, co-authored 34 publications receiving 3385 citations. Previous affiliations of Russell M. Lebovitz include Thomas Jefferson University.
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Journal ArticleDOI
Neurodegeneration, myocardial injury, and perinatal death in mitochondrial superoxide dismutase-deficient mice.
Russell M. Lebovitz,Heju Zhang,Hannes Vogel,Joiner Cartwright,Lianna Dionne,Naifang Lu,Shiu Huang,Martin M. Matzuk +7 more
TL;DR: Observations indicate that SOD2 deficiency causes increased susceptibility to oxidative mitochondrial injury in central nervous system neurons, cardiac myocytes, and other metabolically active tissues after postnatal exposure to ambient oxygen concentrations.
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Growth retardation and cysteine deficiency in gamma-glutamyl transpeptidase-deficient mice
Michael W. Lieberman,Amy L. Wiseman,Zheng-Zheng Shi,Bing Z. Carter,Roberto Barrios,Ching Nan Ou,Patricia Chévez-Barrios,Yibin Wang,Geetha M. Habib,Jerry C. Goodman,Shiu L. Huang,Russell M. Lebovitz,Martin M. Matzuk +12 more
TL;DR: Oral administration of N-acetylcysteine to GGTm1/GGTm1 mice results in normal growth rates and partially restores the normal agouti coat color, demonstrating the importance of GGT and the gamma-glutamyl cycle in cysteine and GSH homeostasis.
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The rat probasin gene promoter directs hormonally and developmentally regulated expression of a heterologous gene specifically to the prostate in transgenic mice.
Norman M. Greenberg,Francesco J. DeMayo,Patricia C. Sheppard,Roberto Barrios,Russell M. Lebovitz,Milton J. Finegold,R. Angelopoulou,R. Angelopoulou,Janice G. Dodd,M. L. Duckworth,Jeffrey M. Rosen,Robert J. Matusik +11 more
TL;DR: These studies demonstrate that a minimal rat probasin promoter can target heterologous gene expression specifically to the prostate in a developmentally and hormonally regulated fashion.
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Ovarian function in superoxide dismutase 1 and 2 knockout mice.
TL;DR: It is demonstrated that female homozygous mutant mice lacking SOD1 can survive to the adult stage but are subfertile and suggested that SOD2 is not essential for ovarian function, and an important role of S OD1 in female reproductive function is supported.
Journal ArticleDOI
Manganese Superoxide Dismutase Protects nNOS Neurons from NMDA and Nitric Oxide-Mediated Neurotoxicity
Mirella Gonzalez-Zulueta,Lisa M. Ensz,Galina Mukhina,Russell M. Lebovitz,Ralf M. Zwacka,John F. Engelhardt,Larry W. Oberley,Valina L. Dawson,Ted M. Dawson +8 more
TL;DR: MnSOD is a major protective protein that appears to be essential for the resistance of nNOS neurons in cortical cultures to NMDA mediated neurotoxicity.