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Sara Trifari

Researcher at Genentech

Publications -  17
Citations -  3344

Sara Trifari is an academic researcher from Genentech. The author has contributed to research in topics: T cell & Genetic enhancement. The author has an hindex of 11, co-authored 17 publications receiving 3117 citations. Previous affiliations of Sara Trifari include La Jolla Institute for Allergy and Immunology.

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Human IL-25- and IL-33-responsive type 2 innate lymphoid cells are defined by expression of CRTH2 and CD161.

TL;DR: This work describes another lineage-negative CD127+CD161+ ILC population found in humans that expressed the chemoattractant receptor CRTH2 and identifies a unique type of human ILC that provides an innate source of T helper type 2 (TH2) cytokines.
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Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from T H -17, T H 1 and T H 2 cells

TL;DR: A previously uncharacterized IL-22-producing human helper T cell population that coexpressed the chemokine receptor CCR6 and the skin-homing receptors CCR4 and CCR10 is described, which may be important in skin homeostasis and pathology.
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Human NKp44+IL-22+ cells and LTi-like cells constitute a stable RORC+ lineage distinct from conventional natural killer cells

TL;DR: It is proposed that CD127+RORC+ cells, although they share some characteristics with cNK cells, represent a functionally and developmentally distinct lineage.
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Regulation of cytokine secretion in human CD127(+) LTi-like innate lymphoid cells by Toll-like receptor 2.

TL;DR: The results indicate that human LTi-like ILC can directly sense bacterial components and unravel a previously unrecognized functional heterogeneity among this important population of innate lymphoid cells.
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Wiskott-Aldrich syndrome protein regulates lipid raft dynamics during immunological synapse formation.

TL;DR: It is demonstrated that the Wiskott-Aldrich syndrome protein (WASP) is recruited to lipid rafts immediately after TCR and CD28 triggering and is required for the movements of lipid rafting.