S
Seiji Hori
Researcher at Kyoto University
Publications - 22
Citations - 5846
Seiji Hori is an academic researcher from Kyoto University. The author has contributed to research in topics: Valosin-containing protein & Endothelin receptor. The author has an hindex of 18, co-authored 21 publications receiving 5672 citations. Previous affiliations of Seiji Hori include Osaka Bioscience Institute.
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Journal ArticleDOI
Cloning and expression of a cDNA encoding an endothelin receptor
TL;DR: The cloning of a complementary DNA encoding a bovine endothelin receptor is reported, which has a transmembrane topology similar to that of other G protein-coupled receptors and shows specific binding, with the highest selectivity to ET-1 in animal cells trans-fected with the cloned cDNA.
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ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats
Hideki Nishitoh,Atsushi Matsuzawa,Kei Tobiume,Kaoru Saegusa,Kohsuke Takeda,Kiyoshi Inoue,Seiji Hori,Seiji Hori,Akira Kakizuka,Akira Kakizuka,Hidenori Ichijo +10 more
TL;DR: It is suggested that ASK1 is a key element in ER stress-induced cell death that plays an important role in the neuropathological alterations in polyQ diseases.
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Molecular characterization of a new metabotropic glutamate receptor mGluR7 coupled to inhibitory cyclic AMP signal transduction.
Naoyuki Okamoto,Seiji Hori,Chihiro Akazawa,Yasunori Hayashi,Ryuichi Shigemoto,Nobuhiro Mizuno,Shigetada Nakanishi +6 more
TL;DR: RNA blot and in situ hybridization analyses of mGLUR7 mRNA indicated that it is widely expressed in many neuronal cells of the central nervous system and is thus different from the more limitedly expressed mGluR4 or mGLuR6 mRNA.
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Distinct tissue distribution and cellular localization of two messenger ribonucleic acids encoding different subtypes of rat endothelin receptors
TL;DR: It is demonstrated that the mRNAs for the two subtypes of rat ET receptors show specialized expression patterns of cell types in both brain and peripheral tissues.
Journal Article
Tumor Growth Inhibition by Arsenic Trioxide (As2O3) in the Orthotopic Metastasis Model of Androgen-independent Prostate Cancer
Hiroshi Maeda,Seiji Hori,Hideki Nishitoh,Hidenori Ichijo,Osamu Ogawa,Yoshiyuki Kakehi,Akira Kakizuka +6 more
TL;DR: It is established that As2O3 provides a novel, safe approach for treatment of androgen-independent prostate cancer and Generation of ROS as a therapeutic target for the potentiation of As 2O3-induced apoptosis also was shown.