S
Songhee Jeon
Researcher at Chonnam National University
Publications - 61
Citations - 1183
Songhee Jeon is an academic researcher from Chonnam National University. The author has contributed to research in topics: Neuroprotection & Parkinson's disease. The author has an hindex of 17, co-authored 58 publications receiving 973 citations. Previous affiliations of Songhee Jeon include Dongguk University.
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Electromagnetic fields induce neural differentiation of human bone marrow derived mesenchymal stem cells via ROS mediated EGFR activation
TL;DR: EMF induce neural differentiation through activation of EGFR signaling and mild generation of ROS in BM-MSCs by examining the signaling pathways involved in the neural differentiation by EMF.
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Proteomic analysis of the neuroprotective mechanisms of acupuncture treatment in a Parkinson's disease mouse model
Songhee Jeon,Youn-Jung Kim,Seung-Tae Kim,Woongjoon Moon,Younbyoung Chae,Minjeong Kang,Mi-Young Chung,Hyangsook Lee,Mi-Sook Hong,Joo-Ho Chung,Tong H. Joh,Hyejung Lee,Hi-Joon Park +12 more
TL;DR: Results suggest that acupoint GB34‐specific EA changes protein expression profiles in the SN in favor of DA neuronal survival in MPTP‐treated mice, and that EA treatment may be an effective therapy for PD patients.
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From Peripheral to Central: The Role of ERK Signaling Pathway in Acupuncture Analgesia
Ji Yeun Park,Jongbae Park,Songhee Jeon,Ah Reum Doo,Seung-Nam Kim,Hyangsook Lee,Younbyoung Chae,William Maixner,Hyejung Lee,Hi-Joon Park,Hi-Joon Park +10 more
TL;DR: The novel evidence of the local molecular signaling in acupuncture analgesia is presented by demonstrating that ERK activation in the skin layer contributes to the analgesic effect of acupuncture in a mouse pain model, which improves the understanding of the scientific basis underlyingupuncture analgesia.
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Acupuncture accelerates wound healing in burn-injured mice
TL;DR: It is demonstrated that acupuncture accelerates the skin regeneration process following deep second degree burns.
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Inhibition of JNK/dFOXO pathway and caspases rescues neurological impairments in Drosophila Alzheimer's disease model.
Yoon Ki Hong,Soojin Lee,Seung Hwan Park,Jang Ho Lee,Seung Yeop Han,Sang Tae Kim,Young-Kyoon Kim,Songhee Jeon,Byung-Soo Koo,Kyoung Sang Cho +9 more
TL;DR: JNK activity was increased in A β42-expressing brains, and the Aβ42-induced defects were rescued by reducing JNK or caspase activity through genetic modification or pharmacological treatment, suggesting that the JNK/dFOXO pathway confers a therapeutic potential for AD.