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Stephan F. Taylor

Researcher at University of Michigan

Publications -  201
Citations -  18385

Stephan F. Taylor is an academic researcher from University of Michigan. The author has contributed to research in topics: Cognition & Prefrontal cortex. The author has an hindex of 58, co-authored 179 publications receiving 16611 citations. Previous affiliations of Stephan F. Taylor include United States Department of Veterans Affairs & Veterans Health Administration.

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Updating Beliefs for a Decision: Neural Correlates of Uncertainty and Underconfidence

TL;DR: Examination of neural activity while subjects accumulated sequential pieces of evidence and then made a decision suggests that neural mechanisms of uncertainty depend on the stage of decision-making and that greater subjective uncertainty when evaluating evidence is associated with activity in ventromedial brain regions, even in the absence of overt risk.
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μ-Opioid receptors and limbic responses to aversive emotional stimuli

TL;DR: Higher baseline μ-opioid receptor binding potential was associated with lower regional cerebral blood flow in this region during presentation of emotionally salient stimuli, consistent with an inhibitory/anxiolytic role of the endogenous opioid system in limbic regions of the temporal lobe and basal forebrain.
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Vesicular monoamine transporter concentrations in bipolar disorder type I, schizophrenia, and healthy subjects

TL;DR: The patterns of regional VMAT2 expression, and by extension, the concentration of monoaminergic synaptic terminals, differ between BDI, SCH, and a control group, and may relate to both similarities and differences in the presentation or clinical course of these syndromes.
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Phasic and enduring negative symptoms in schizophrenia: biological markers and relationship to outcome.

TL;DR: Comparisons of correlates of enduring and phasic negative symptoms in drug-free schizophrenic patients suggest that acute-phase (phasic) negative symptoms may be pathophysiologically distinct from enduring negative symptoms that persist through the residual phase.
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Chronic medication does not affect hyperactive error responses in obsessive-compulsive disorder

TL;DR: The finding of greater ERNs in OCD patients than in controls, irrespective of medication use, confirms previous findings of an exaggerated error response in OCD and suggests that elevated error signals in OCD may be disorder-specific.