scispace - formally typeset
R

Robert C. Welsh

Researcher at University of Utah

Publications -  189
Citations -  12408

Robert C. Welsh is an academic researcher from University of Utah. The author has contributed to research in topics: Default mode network & Prefrontal cortex. The author has an hindex of 62, co-authored 173 publications receiving 11202 citations. Previous affiliations of Robert C. Welsh include Icahn School of Medicine at Mount Sinai & Johns Hopkins University.

Papers
More filters
Journal ArticleDOI

Aging and the Neural Correlates of Successful Picture Encoding: Frontal Activations Compensate for Decreased Medial-Temporal Activity

TL;DR: Detailed fMRI analyses suggest that prefrontal regions could serve a compensatory role for declines in medial-temporal activations with age, and correlations between inferior frontal and parahippocampal activity were significantly negative for old but not young.
Journal ArticleDOI

Diffusion tensor imaging of the brain: review of clinical applications.

TL;DR: The theoretical background to diffusion tensor imaging and its commoner clinical applications, such as cerebral ischemia, brain maturation and traumatic brain injury, and its potential use in diseases such as epilepsy, multiple sclerosis, and Alzheimer's disease are reviewed.
Journal ArticleDOI

Neural correlates of individual ratings of emotional salience: a trial-related fMRI study

TL;DR: The neural basis for these components of endogenous salience during such appraisals of emotional stimuli is examined using trial-related functional magnetic resonance imaging (fMRI).
Journal ArticleDOI

Error-related hyperactivity of the anterior cingulate cortex in obsessive-compulsive disorder.

TL;DR: Error-processing abnormalities within the rostral anterior cingulate occur in the absence of symptom expression in patients with OCD.
Journal ArticleDOI

Neural dysregulation in posttraumatic stress disorder: evidence for disrupted equilibrium between salience and default mode brain networks.

TL;DR: A relative dominance of threat-sensitive circuitry in PTSD is suggested, even in task-free conditions, and Disequilibrium between large-scale networks subserving salience detection versus internally focused thought may be associated with PTSD pathophysiology.