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Stephania A. Cormier
Researcher at Louisiana State University
Publications - 114
Citations - 5186
Stephania A. Cormier is an academic researcher from Louisiana State University. The author has contributed to research in topics: Immune system & Inflammation. The author has an hindex of 36, co-authored 103 publications receiving 4347 citations. Previous affiliations of Stephania A. Cormier include University of California, Los Angeles & Mayo Clinic.
Papers
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Journal ArticleDOI
Defining a link with asthma in mice congenitally deficient in eosinophils.
James J. Lee,Dawn Dimina,Mi Mi P. Macias,Sergei I. Ochkur,Michael P. McGarry,K.R. O'Neill,Cheryl A. Protheroe,R.S. Pero,Thanh H Nguyen,Stephania A. Cormier,Stephania A. Cormier,Elizabeth Lenkiewicz,Dana Colbert,Lisa Rinaldi,Steven J. Ackerman,Charles G. Irvin,Nancy A. Lee +16 more
TL;DR: The development of an eosinophil-less mouse now permits an unambiguous assessment of a number of human diseases that have been linked to this granulocyte, including allergic diseases, parasite infections, and tumorigenesis.
Journal ArticleDOI
Copper oxide nanoparticles induce oxidative stress and cytotoxicity in airway epithelial cells
Baher Fahmy,Stephania A. Cormier +1 more
TL;DR: There is a high degree of variability in the cytotoxic effects of metal oxides, this variability is not due to the solubility of the transition metal, and that this variability appears to involve sustained oxidative stress possibly due to redox cycling.
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Exposure to electronic cigarettes impairs pulmonary anti-bacterial and anti-viral defenses in a mouse model
Thomas E. Sussan,Sachin Gajghate,Rajesh K. Thimmulappa,Jinfang Ma,Jung Hyun Kim,Kuladeep Sudini,Nicola Consolini,Stephania A. Cormier,Slawo Lomnicki,Farhana Hasan,Andrew Pekosz,Shyam Biswal +11 more
TL;DR: It is demonstrated that E-cig exposure elicits impaired pulmonary anti-microbial defenses and must be rigorously tested in users for their effects on immune response and susceptibility to bacterial and viral infections.
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Origin and health impacts of emissions of toxic by-products and fine particles from combustion and thermal treatment of hazardous wastes and materials.
TL;DR: Fine PM and ultrafine PM are effective delivery agents for PAHs, CHCs, and toxic metals and the origin of each of these classes of pollutants, the nature of their association with combustion-generated PM, and the mechanisms of their known and potential health impacts are discussed.
Journal ArticleDOI
Pivotal Advance: eosinophil infiltration of solid tumors is an early and persistent inflammatory host response
Stephania A. Cormier,Anna G. Taranova,Carrie E. Bedient,Thanh H Nguyen,Cheryl A. Protheroe,R.S. Pero,Dawn Dimina,Sergei I. Ochkur,K.R. O'Neill,Dana Colbert,Theresa R. Lombari,Stephanie L. Constant,Michael P. McGarry,James J. Lee,Nancy A. Lee +14 more
TL;DR: It is demonstrated that the infiltration of tumors by eosinophils is an early and persistent response that is spatial‐restricted, suggesting that eos inophils are part of an early inflammatory reaction at the site of tumorigenesis.