T
Thomas Reimann
Researcher at Dresden University of Technology
Publications - 44
Citations - 920
Thomas Reimann is an academic researcher from Dresden University of Technology. The author has contributed to research in topics: Aquifer & MODFLOW. The author has an hindex of 15, co-authored 35 publications receiving 759 citations. Previous affiliations of Thomas Reimann include Collège de France & Helmholtz-Zentrum Dresden-Rossendorf.
Papers
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Journal ArticleDOI
Loss of caveolin expression in type I pneumocytes as an indicator of subcellular alterations during lung fibrogenesis.
Michael Kasper,Thomas Reimann,Ute Hempel,K.-W. Wenzel,A Bierhaus,D Schuh,V. Dimmer,Gunter Haroske,Martin Müller +8 more
TL;DR: It is demonstrated that caveolin expression in the alveolar epithelium of rats and mini pigs is strikingly downregulated after irradiation-induced lung injury.
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Transforming growth factor-β1 induces activation of Ras, Raf-1, MEK and MAPK in rat hepatic stellate cells
Thomas Reimann,Ute Hempel,Stefan Krautwald,Andreas Axmann,Roland Scheibe,Dagmar Seidel,K.-W. Wenzel +6 more
TL;DR: It is suggested that the use of nanofiltration membranes for the recovery of phosphorous with a second type of technology is a viable process and should be considered as a possibility for further research.
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Anisotropic field-induced ordering in the triangular-lattice quantum spin liquid NaYbSe 2
K. M. Ranjith,S. Luther,S. Luther,Thomas Reimann,Burkhard Schmidt,Ph. Schlender,Jörg Sichelschmidt,H. Yasuoka,André M. Strydom,Y. Skourski,J. Wosnitza,J. Wosnitza,H. Kühne,Th. Doert,Michael Baenitz +14 more
TL;DR: In this article, the authors derived the highly anisotropic magnetic phase diagram, which is discussed in comparison with the existing theoretical models for spin-$\frac{1}{2}$ triangular-lattice antiferromagnets.
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Effects of dynamically variable saturation and matrix-conduit coupling of flow in karst aquifers
TL;DR: In this paper, a new karst model (ModBraC) is presented that accounts for unsteady and non-uniform discrete flow in variably saturated conduits employing the Saint-Venant equations.
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Transforming growth factor-beta1-induced activation of the Raf-MEK-MAPK signaling pathway in rat lung fibroblasts via a PKC-dependent mechanism.
TL;DR: It is shown that calcium-dependent isoforms of PKC are required for MAPK activation by TGF-beta1, which regulates cell proliferation and turnover of macromolecular components of the extracellular matrix.