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Ulrich T. Hopt

Researcher at University of Freiburg

Publications -  349
Citations -  11279

Ulrich T. Hopt is an academic researcher from University of Freiburg. The author has contributed to research in topics: Pancreatitis & Pancreas. The author has an hindex of 50, co-authored 347 publications receiving 10112 citations. Previous affiliations of Ulrich T. Hopt include University Medical Center Freiburg.

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Network of surgical wound centers using a new electronic data processing documentation system

TL;DR: A network of ten wound care centers was created and data were documented in a new computerized wound documentation system, which was integrated into the clinical routine and enables clear and standardized documentation.
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Organ-specific monocyte activation in necrotizing pancreatitis in mice

TL;DR: Although inflammatory mediators increased during 24 h after pancreatitis induction, monocyte activation lasted for at least 48 h, and the latter is not limited to blood but also detected in isolated liver and spleen monocytes.
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Biliary stenting of an iatrogenic esophageal perforation following corrosive esophagitis in a 5-year-old child

TL;DR: Owing to a lack of pediatric treatment options for severe sepsis in combination with a pronounced esophageal stricture, the perforation was sealed with a partially covered biliary stent, which allowed the esophagus to function until definitive repair could be attempted as primary repair was not possible.
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Hyperacute Rejection of a Living Unrelated Kidney Graft

TL;DR: The case report of a 59-year-old man, who received a blood group identical living unrelated kidney graft, shows signs of hyperacute rejection and had to be removed, and recommends examining more closely recipients of second allografts, considering not only a positive T-cell crossmatch but also a positive B-cellCrossmatch as exclusion criteria for transplantation.
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Effect of liver regeneration after partial hepatectomy and ischemia-reperfusion on expression of growth factor receptors.

TL;DR: At least partially due to a post-transcriptional process, there is a nadir in the expression of the analysed receptors 24 h after liver injury, suggesting that a therapeutic use of growth factors to stimulate liver regeneration 24H after the damage might be not successful.