U
Ulrich T. Hopt
Researcher at University of Freiburg
Publications - 349
Citations - 11279
Ulrich T. Hopt is an academic researcher from University of Freiburg. The author has contributed to research in topics: Pancreatitis & Pancreas. The author has an hindex of 50, co-authored 347 publications receiving 10112 citations. Previous affiliations of Ulrich T. Hopt include University Medical Center Freiburg.
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Network of surgical wound centers using a new electronic data processing documentation system
TL;DR: A network of ten wound care centers was created and data were documented in a new computerized wound documentation system, which was integrated into the clinical routine and enables clear and standardized documentation.
Journal ArticleDOI
Organ-specific monocyte activation in necrotizing pancreatitis in mice
Andrea I. Schmidt,Gabriel Seifert,Robert Lauch,Guido Wolff-Vorbeck,Sophia Chikhladze,Ulrich T. Hopt,Uwe A. Wittel +6 more
TL;DR: Although inflammatory mediators increased during 24 h after pancreatitis induction, monocyte activation lasted for at least 48 h, and the latter is not limited to blood but also detected in isolated liver and spleen monocytes.
Journal ArticleDOI
Biliary stenting of an iatrogenic esophageal perforation following corrosive esophagitis in a 5-year-old child
TL;DR: Owing to a lack of pediatric treatment options for severe sepsis in combination with a pronounced esophageal stricture, the perforation was sealed with a partially covered biliary stent, which allowed the esophagus to function until definitive repair could be attempted as primary repair was not possible.
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Hyperacute Rejection of a Living Unrelated Kidney Graft
D. Tittelbach-Helmrich,Dirk Bausch,Oliver Drognitz,Heike Goebel,Christian Schulz-Huotari,Albrecht Kramer-Zucker,Ulrich T. Hopt,Przemyslaw Pisarski +7 more
TL;DR: The case report of a 59-year-old man, who received a blood group identical living unrelated kidney graft, shows signs of hyperacute rejection and had to be removed, and recommends examining more closely recipients of second allografts, considering not only a positive T-cell crossmatch but also a positive B-cellCrossmatch as exclusion criteria for transplantation.
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Effect of liver regeneration after partial hepatectomy and ischemia-reperfusion on expression of growth factor receptors.
TL;DR: At least partially due to a post-transcriptional process, there is a nadir in the expression of the analysed receptors 24 h after liver injury, suggesting that a therapeutic use of growth factors to stimulate liver regeneration 24H after the damage might be not successful.