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Winfried März

Researcher at Heidelberg University

Publications -  398
Citations -  46505

Winfried März is an academic researcher from Heidelberg University. The author has contributed to research in topics: Vitamin D and neurology & Genome-wide association study. The author has an hindex of 74, co-authored 361 publications receiving 36423 citations. Previous affiliations of Winfried März include University of Ulm & University of Graz.

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Utilization of lipid-modifying therapy and low-density lipoprotein cholesterol goal attainment in patients at high and very-high cardiovascular risk: Real world evidence from Germany

TL;DR: In this article, the Cegedim Longitudinal practice database in Germany was used to assess LLT treatment patterns and LDL-C threshold attainment in Germany in a large, real-world cohort of patients.
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The role of vitamin D deficiency in cardiovascular disease: where do we stand in 2013?

TL;DR: More emphasis should be placed on RCTs among severely vitamin D-deficient populations who would most likely benefit from vitamin D treatment, as vitamin D supplementation with doses recommended for osteoporosis treatment is neither proven to be beneficial nor harmful in cardiovascular diseases.
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Vitamin D and Cancer Mortality: Systematic Review of Prospective Epidemiological Studies

TL;DR: The majority of studies in cancer patients showed that patients with higher 25(OH)D levels had a decreased risk of mortality and there was no indication for increased mortality risk with higher vitamin D levels in any cancer cohort.
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Sex steroids and mortality in men referred for coronary angiography

TL;DR: Evaluated whether total testosterone, free testosterone and sex hormone–binding globulin are associated with all‐cause mortality and specific fatal events and accumulating evidence suggests that sex steroids areassociated with various chronic diseases.
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Inflammation-associated graft loss in renal transplant recipients

TL;DR: The inflammation markers hsCRP and IL-6 are associated with long-term graft outcomes in renal transplant recipients and were independently associated with death-censored graft loss, the composite end points graft Loss or death and doubling of serum creatinine, graft loss or death.