scispace - formally typeset
Y

Yoichi Takami

Researcher at Osaka University

Publications -  88
Citations -  2582

Yoichi Takami is an academic researcher from Osaka University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 24, co-authored 67 publications receiving 2122 citations. Previous affiliations of Yoichi Takami include Asahikawa Medical University & University of California, San Francisco.

Papers
More filters
Journal ArticleDOI

Novel Autologous Cell Therapy in Ischemic Limb Disease Through Growth Factor Secretion by Cultured Adipose Tissue–Derived Stromal Cells

TL;DR: These adipose tissue–derived cells demonstrated potential asAngiogenic cell therapy for ischemic disease, which appears to be mainly achieved by their ability to secrete angiogenic growth factors.
Journal ArticleDOI

Effects of the creation of arteriovenous fistula for hemodialysis on cardiac function and natriuretic peptide levels in CRF.

TL;DR: It is indicated that creation of an AV fistula has significant effects on cardiac systolic and diastolic performance, and ANP release is induced by volume loading, but BNP release is stimulated by LV diastolics dysfunction.
Journal ArticleDOI

Diagnostic and prognostic value of plasma brain natriuretic peptide in non—dialysis-dependent CRF

TL;DR: Findings support plasma BNP level as a reliable marker of LV overload, even in nondialysis patients with CRF, and suggest that a high B NP level may have powerful predictive potential for heart failure in these patients.
Journal ArticleDOI

Modified forelimb grip strength test detects aging-associated physiological decline in skeletal muscle function in male mice.

TL;DR: The modified forelimb grip strength test, with its improved validity and reliability may be an ideal substitute for the conventional method in detecting skeletal muscle dysfunction in young male dystrophic mice.
Journal ArticleDOI

The Myeloid Transcription Factor KLF2 Regulates the Host Response to Polymicrobial Infection and Endotoxic Shock

TL;DR: In this paper, the authors identify the Kruppel-like transcription factor 2 (KLF2) as a potent regulator of myeloid cell activation in vivo, and demonstrate that exposure to either hypoxia and/or bacterial products reduced KLF2 expression while inducing hypoxyia inducible factor 1α (HIF-1α), findings that were recapitulated in human septic patients.