Example of Drug Metabolism Reviews format
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Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format
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Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format Example of Drug Metabolism Reviews format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access
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Drug Metabolism Reviews — Template for authors

Publisher: Taylor and Francis
Categories Rank Trend in last 3 yrs
Pharmacology, Toxicology and Pharmaceutics (all) #2 of 67 down down by 1 rank
Pharmacology (medical) #25 of 246 down down by 10 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 105 Published Papers | 759 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 22/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.956

16% from 2018

Impact factor for Drug Metabolism Reviews from 2016 - 2019
Year Value
2019 3.956
2018 4.702
2017 4.45
2016 4.097
graph view Graph view
table view Table view

7.2

9% from 2019

CiteRatio for Drug Metabolism Reviews from 2016 - 2020
Year Value
2020 7.2
2019 6.6
2018 7.1
2017 8.3
2016 6.9
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 16% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 9% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.023

9% from 2019

SJR for Drug Metabolism Reviews from 2016 - 2020
Year Value
2020 1.023
2019 1.119
2018 0.944
2017 1.271
2016 1.129
graph view Graph view
table view Table view

1.448

4% from 2019

SNIP for Drug Metabolism Reviews from 2016 - 2020
Year Value
2020 1.448
2019 1.508
2018 1.207
2017 1.391
2016 1.217
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 9% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 4% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Drug Metabolism Reviews

Guideline source: View

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Taylor and Francis

Drug Metabolism Reviews

Drug Metabolism Reviews consistently provides critically needed reviews of an impressive array of drug metabolism research-covering established, new, and potential drugs; environmentally toxic chemicals; absorption; metabolism and excretion; and enzymology of all living specie...... Read More

Pharmacology, Toxicology and Pharmaceutics

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Last updated on
22 Jun 2020
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ISSN
0360-2532
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Impact Factor
High - 2.003
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Taylor and Francis Custom Citation
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys Rev B. 1982; 25(7):4515–4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

Journal Article DOI: 10.3109/03602539709037591
Human Cytochrome P450 Enzymes: A Status Report Summarizing Their Reactions, Substrates, Inducers, and Inhibitors
Slobodan Rendic1, Frederick J. Di Carlo2
01 Feb 1997 - Drug Metabolism Reviews

Abstract:

(1997). Human Cytochrome P450 Enzymes: A Status Report Summarizing Their Reactions, Substrates, Inducers, and Inhibitors. Drug Metabolism Reviews: Vol. 29, No. 1-2, pp. 413-580. (1997). Human Cytochrome P450 Enzymes: A Status Report Summarizing Their Reactions, Substrates, Inducers, and Inhibitors. Drug Metabolism Reviews: Vol. 29, No. 1-2, pp. 413-580. read more read less

Topics:

CYP2B6 (59%)59% related to the paper, CYP1A2 (55%)55% related to the paper, Cytochrome P450 (53%)53% related to the paper, Drug metabolism (50%)50% related to the paper
1,170 Citations
Journal Article DOI: 10.1080/03602530600971974
Mechanistic Studies of the Nrf2-Keap1 Signaling Pathway
Donna D. Zhang1
01 Jan 2006 - Drug Metabolism Reviews

Abstract:

Since eukaryotic cells constantly encounter various environmental insults, they have evolved defense mechanisms to cope with toxicant- and carcinogen-induced oxidative stress or electrophiles. One of the most important cellular defense mechanisms against oxidative stress or electrophiles is mediated by the transcription facto... Since eukaryotic cells constantly encounter various environmental insults, they have evolved defense mechanisms to cope with toxicant- and carcinogen-induced oxidative stress or electrophiles. One of the most important cellular defense mechanisms against oxidative stress or electrophiles is mediated by the transcription factor Nrf2. Under the basal condition, Nrf2-dependent transcription is repressed by a negative regulator Keap1. When cells are exposed to oxidative stress, electrophiles, or chemopreventive agents, Nrf2 escapes Keap1-mediated repression and activates antioxidant responsive element (ARE)-dependent gene expression to maintain cellular redox homeostasis. Beyond its antioxidant function, Nrf2 has recently been recognized as a key factor regulating an array of genes that defend cells against the deleterious effects of environmental insults. Since this Nrf2-dependent cellular defense response is able to protect multi-organs or multi-tissues, activation of Nrf2 has been implicated in conferring ... read more read less

Topics:

Cellular defense response (63%)63% related to the paper, KEAP1 (52%)52% related to the paper, Transcription factor (50%)50% related to the paper
952 Citations
Journal Article DOI: 10.3109/03602539808996310
Reactive oxygen-mediated protein oxidation in aging and disease.
Earl R. Stadtman1, Barbara S. Berlett1
01 May 1998 - Drug Metabolism Reviews

Abstract:

Highly reactive oxygen species that are formed during normal metabolism and under conditions of oxidative stress are able to oxidize proteins or convert lipid and carbohydrate derivatives to compounds that react with functional groups on proteins. Among other changes, these ROS-mediated reactions lead to the formation of prot... Highly reactive oxygen species that are formed during normal metabolism and under conditions of oxidative stress are able to oxidize proteins or convert lipid and carbohydrate derivatives to compounds that react with functional groups on proteins. Among other changes, these ROS-mediated reactions lead to the formation of protein carbonyl derivatives, which serves as a marker of ROS-mediated protein damage. On the basis of this marker, it is established that oxidatively damaged protein is associated with aging and some diseases. The accumulation of oxidatively damaged protein reflects the balance among a myriad of factors that govern the rates of ROS generation and the rate at which damaged protein is degraded. Peroxynitrite, which is formed under normal physiological conditions, is able to oxidize methionine residues in proteins and to nitrate tyrosine residues; however, its ability to do so is dependent on the availability of CO2, which stimulates the nitration of tyrosine residues but inhibits the oxidation of methionine residues. Nitration of tyrosine residues may contribute to peroxynitrite toxicity, as nitration precludes the phosphorylation or nucleotidylation of tyrosine residues and thereby seriously compromises one of the most important mechanisms of cellular regulation and signal transduction. read more read less

Topics:

Protein oxidation (59%)59% related to the paper, Tyrosine (55%)55% related to the paper, Peroxynitrite (54%)54% related to the paper, Phosphorylation (53%)53% related to the paper, Oxidative stress (52%)52% related to the paper
863 Citations
Journal Article DOI: 10.1081/DMR-120001392
Summary of information on human cyp enzymes: human p450 metabolism data
Slobodan Rendic1
15 Apr 2002 - Drug Metabolism Reviews

Abstract:

This chapter is an update of the data on substrates, reactions, inducers, and inhibitors of human CYP enzymes published previously by Rendic and DiCarlo (1), now covering selection of the literature through 2001 in the reference section. The data are presented in a tabular form (Table 1) to provide a framework for predicting ... This chapter is an update of the data on substrates, reactions, inducers, and inhibitors of human CYP enzymes published previously by Rendic and DiCarlo (1), now covering selection of the literature through 2001 in the reference section. The data are presented in a tabular form (Table 1) to provide a framework for predicting and interpreting the new P450 metabolic data. The data are formatted in an Excel format as most suitable for off-line searching and management of the Web-database. The data are presented as stated by the author(s) and in the case when several references are cited the data are presented according to the latest published information. The searchable database is available either as an Excel file (for information contact the author), or as a Web-searchable database (Human P450 Metabolism Database, www.gentest.com) enabling the readers easy and quick approach to the latest updates on human CYP metabolic reactions. read more read less
788 Citations
Journal Article DOI: 10.1081/DMR-100102336
Quantification and significance of protein oxidation in biological samples
Emily Shacter1
10 Oct 2000 - Drug Metabolism Reviews

Abstract:

Protein oxidation is defined here as the covalent modification of a protein induced either directly by reactive oxygen species or indirectly by reaction with secondary by-products of oxidative stress. Oxidative modification of proteins can be induced experimentally by a wide array of prooxidant agents and occurs in vivo durin... Protein oxidation is defined here as the covalent modification of a protein induced either directly by reactive oxygen species or indirectly by reaction with secondary by-products of oxidative stress. Oxidative modification of proteins can be induced experimentally by a wide array of prooxidant agents and occurs in vivo during aging and in certain disease conditions. Oxidative changes to proteins can lead to diverse functional consequences, such as inhibition of enzymatic and binding activities, increased susceptibility to aggregation and proteolysis, increased or decreased uptake by cells, and altered immunogenicity. There are numerous types of protein oxidative modification and these can be measured with a variety of methods. Protein oxidation serves as a useful marker for assessing oxidative stress in vivo. There are both advantages and disadvantages to using proteins for this purpose compared to lipids and DNA. Finally, it is important to monitor the degree of oxidative modification of therapeutic proteins manufactured for commercial use. This review will examine various aspects of protein oxidation, with emphasis on using proteins as markers of oxidative stress in biological samples. read more read less

Topics:

Protein oxidation (67%)67% related to the paper, Oxidative stress (57%)57% related to the paper, Oxidative phosphorylation (52%)52% related to the paper, Reactive oxygen species (50%)50% related to the paper
761 Citations
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Frequently asked questions

1. Can I write Drug Metabolism Reviews in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Drug Metabolism Reviews guidelines and auto format it.

2. Do you follow the Drug Metabolism Reviews guidelines?

Yes, the template is compliant with the Drug Metabolism Reviews guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Drug Metabolism Reviews?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Drug Metabolism Reviews citation style.

4. Can I use the Drug Metabolism Reviews templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Drug Metabolism Reviews.

5. Can I use a manuscript in Drug Metabolism Reviews that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Drug Metabolism Reviews that you can download at the end.

6. How long does it usually take you to format my papers in Drug Metabolism Reviews?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Drug Metabolism Reviews.

7. Where can I find the template for the Drug Metabolism Reviews?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Drug Metabolism Reviews's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Drug Metabolism Reviews's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Drug Metabolism Reviews an online tool or is there a desktop version?

SciSpace's Drug Metabolism Reviews is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Drug Metabolism Reviews?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Drug Metabolism Reviews?”

11. What is the output that I would get after using Drug Metabolism Reviews?

After writing your paper autoformatting in Drug Metabolism Reviews, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Drug Metabolism Reviews's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Drug Metabolism Reviews?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Drug Metabolism Reviews. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Drug Metabolism Reviews?

The 5 most common citation types in order of usage for Drug Metabolism Reviews are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Drug Metabolism Reviews?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Drug Metabolism Reviews's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Drug Metabolism Reviews in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Drug Metabolism Reviews Endnote style according to Elsevier guidelines.

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