Showing papers in "Journal of Critical Care in 1992"

TL;DR: In conclusion, it is very unlikely that volume loading will increase the cardiac output in a patient who does not have an inspiratory fall in Pra, whereasVolume loading will usually increase the heart output of patients who have an inspiration fall in pra.

TL;DR: It is concluded that in this model, high-volume venovenous hemofiltration significantly improves cardiac function but does not affect pulmonary vascular resistance or systemic vascular resistance, while the use of the extracorporeal circuit adversely affects cardiac function.

TL;DR: The bedside determination of the ΔPpao/ΔPpa ratio should identify the cases in which Ppao measurements are not valid more reliably than lateral chest x-ray film, thus avoiding misleading interpretations of P pao during PEEP ventilation.

TL;DR: The analysis indicates that the incidences of pneumonia and atelectasis were significantly reduced by KT tables, and KT did not appear to have a statistically significant effect on adult respiratory distress syndrome, pressure ulcers, embolisms, mortality, or length of hospital stay.

TL;DR: The hypothesis that rapidly expanding AV pH and AV Pco2 represent metabolic acidosis and that these parameters may potentially be used to detect dysoxia is supported.

TL;DR: The oxygen demands of the patient with ARDS should be minimized to lower the needs for ventilation and pressure, and the least mean alveolar pressure necessary to accomplish unequivocal therapeutic goals should be used.

TL;DR: Patients with coronary ischemia appear to benefit from mechanical ventilation, although patient selection and the mechanisms involved need much more investigation and positive end-expiratory pressure-induced changes in abdominal pressure at different blood volume levels need to be considered.

TL;DR: The concept of local pulmonary production of this cytokine in ARDS is supported and production of TNF did not vary over the study period and was observed not only in patients with septic syndrome but also in patients without sepsis.

TL;DR: The assumptions necessary for appropriate use of multivariate approaches, as well as discussion of the interpretations to be drawn from the information provided by computer programs, are presented.

TL;DR: The oxygen consumption/ delivery relationship (VO2/602) was studied in 18 sedated, paralyzed, septic adult respiratory distress syndrome patients and found changes in DO2 decreased with PEEP, while oxygen extraction ratio and arterial-mixed venous oxygen difference increased as PEEP was applied.

TL;DR: The agents reviewed in this article have the potential to ameliorate or prevent cellular and organ dysfunction and well-controlled and designed studies that modify their action should be undertaken in multi-institutional settings.

TL;DR: The mechanisms that cause flow, volume, and time dependence of Rrs in normal subjects and in patients with ARDS and COPD are reviewed.

TL;DR: The high values of Wi,rs observed in adult respiratory distress syndrome patients would likely cause inspiratory muscle fatigue during spontaneous breathing, hence necessitating mechanical support.

TL;DR: It is concluded that during resuscitation from hypovolemic shock, correction of acidemia using bicarbonate does not increase left ventricular contractility.

TL;DR: The purposes of this paper are to review the elements of the pulmonary defense system and the pathogenesis of lower respiratory infections, and to review recent investigational work in which attempts have been made to modulate the immune response of the host and bolster host defenses.

TL;DR: The result of the LFPPV-ECCO2R clinical trial is a potent reminder of the importance of controls in therapy evaluation, and ethical objections to controlled trials of therapies that are not supported by credible data should be viewed with caution.

TL;DR: Measurements of P ao during the whole breathing cycle cannot be assumed to be a valid index of P alv, and it is indicated that measurements of PAlv is feasible using the flow interruption technique.

TL;DR: In this article, balloon filling in the inferior vena cave was used to study the relationship between oxygen uptake (VO2) and oxygen supply (DO2) during an acute reduction in blood flow.

TL;DR: SOD and CAT prevent the systemic leukopenia that accompanies pulmonary artery reperfusion, but do not prevent the injury to the reperfused lung, and the sparing effect on the contralateral lung suggests that the mechanisms of injury for that lung differs from the mechanism of injury toThe occluded lung.

TL;DR: It is determined that LV preload decreases before the transseptal pressure gradient decreases and the septum shifts leftward, and LV distensibility does not appear to be altered by leftward septal shift when it occurs provided the pericardium is opened.

TL;DR: The results indicate that the combination of dobutamine and rtPA may be appropriate therapy when a low CO complicates pulmonary embolism.

TL;DR: It is suggested that enteric GNB rapidly colonize the oropharynx and upper gastrointestinal tract in this model of renal infarction, and that gastric acidity plays a minor role in the pathogenesis of this process.

TL;DR: A prospective study of two different respiratory culture techniques was performed in a group of hospitalized, ventilator-dependent children to compare costs and complications and to determine whether upper airway contamination of respiratory cultures could be reduced.

TL;DR: The mean arterial pressure was found to decrease markedly with the induction of brain death, and this progression occurs despite maximal clinical therapy, including mechanical ventilation, inotropes, and pressor agents.

TL;DR: A method for automatically recording and analyzing expiratory pressure flow curves in mechanically ventilated patients using standard ventilatory equipment and a personal computer is developed and it is concluded that this simple and convenient method allows automatic and accurate construction of pulmonary exhalation flow properties and may enable measurement of passive expiratories resistance.

TL;DR: It is concluded that doxapram can directly augment hypoxic pulmonary vasoconstriction even in the absence of systemic neuronal or humeral input and could act to improve ventilation/ perfusion matching and also to decrease pulmonary edema.