Showing papers in "Physiological and Molecular Plant Pathology in 1999"

TL;DR: SAR, likewise, is a generally occurring phenomenon, that engenders an enhancement of the defensive capacity of plants in response to necrotizing infections, associated with the development of systemic acquired resistance (SAR) against further infection by fungi, bacteria and viruses.

TL;DR: Some of the defenses expressed in plants with induced resistance will be discussed in relation to how the induced plants may restrict disease development.

TL;DR: Clear evidence is provided for reciprocal induced resistance involving certain pathogens and arthropod herbivores of tomato and several insights are provided into the integration and coordination of the induced defenses of tomato against multiple pests.

TL;DR: The results do not support the hypothesis for a strict dichotomy of signaling by insects and pathogens of LOX, PINII and P4 in tomato, but point to a potential vulnerability of acquired resistance evident at the levels of gene expression and response to insect attack.

TL;DR: Toxin accumulation showed a very close relationship with pathogenic changes in host cells, symptom appearance and colonization of host tissues by hyphae, suggesting that the toxins might play an important role in the disease development.

TL;DR: Phytoplasmal infection caused growth inhibition and severe yellowing in both plants, the GY phytoplasma being more virulent than the AP and ASHY agents in periwinkle, with little or no effect caused by either the AP or GY agents.

TL;DR: It is demonstrated that 3-deoxyanthocyanidin phytoalexins represent major components of resistance to C. sublineolum in sorghum.

TL;DR: In this article, immunofluorescence microscopy with two monoclonal antibodies raised to two Trichoderma reesei cellobiohydrolases revealed antigen localization at the germ tube tips of Erysiphe graminis f.sp. hordei.

TL;DR: The ability to accumulate SA was not predictive of the ability to elicit SAR, and SA accumulation alone is not sufficient to elicit the SAR response, as SA accumulated to similar levels during both compatible and incompatible interactions.

TL;DR: The results suggest that MeJA acts by stimulating defence responses of the host plant by inducing the accumulation of free salicylic acid in all parts of the seedlings, whereas bound SA only increased in hypocotyls and cotyledons.

TL;DR: It is proposed that the high concentration of chlorogenic acid present in immature fruit and in fruit from genotypes with high levels of disease resistance may contribute to the brown rot resistance of the tissue by interference with the production of factors involved in degradation of host polymers rather than by direct toxicity to the pathogen.

TL;DR: It is suggested that the resistant vetch defense mechanism involves elevated induction of the phenylproponoid pathway upon Orobanche infection, conferring mechanical and chemical barriers confronting the invading parasite.

TL;DR: A growing body of evidence indicates that most microbial genes conditioning pathogenicity, including the hrp, pth and avr genes, are present because of horizontal gene transfer, often involving movement of large gene clusters on “pathogenicity islands”.

TL;DR: In control leaves, attacked by appressoria from a single inoculation, approximately 67% of penetration attempts successfully formed haustoria, however, in epidermal cells successfully penetrated by the inducer, almost all attacks by the challenger succeeded and accessibility was induced in these cells.

TL;DR: The results suggest that the amount of H2O2 accumulating during an elicitor-induced response in leaves may be sufficient to affect fungal colonization but not to affect viability of host cells unless the Fe2+status in the apoplast is in some way altered by the elicitor to facilitate OH.

TL;DR: A plant’s capacity for the recognition of an incompatible pathogen leads initially to a nearly universal defense reaction, the programmed hypersensitive death of cells in immediate contact with the pathogen.

TL;DR: To investigate early events in the host response to nematode infection, a seedling inoculation procedure was designed that produced hundreds of root tips synchronously infected with Meloidogyne javanica, and RNA extracted was used to produce a cDNA library.

TL;DR: The results indicate that alteration in ethylene perception can markedly reduce the amount of tissue damage during this susceptible response to toxin-induced cell death and suggest strategies for manipulation of cell death to enhance plant disease resistance.

TL;DR: The results suggest that a specialized enzyme system for quickly detoxifying pisatin might be present in most pea pathogens, including N. haematococca PDA genes.

TL;DR: It is assumed that a hypersensitive response was elicited in incompatible and compatible interactions and because of the delay in the induction of the resistance response in the susceptible plant, the virus might be able to spread beyond the necrotic reacting and callose depositing cells thereby establishing a systemic infection.

TL;DR: The system combines biolistic transformation with direct comparison of the fungal attack in transfected and wild type cells and many epidermal cells showed detectable expression of marker genes including a novel improved version of green fluorescent protein (GFP).

TL;DR: Northern and Western blot analyses show that the gene encoding SP1 is expressed and that the protein is secreted during infection of wheat leaves.

TL;DR: A lipase produced by Botrytis cinerea was detected using specific antilipase antibodies during infection of grapevine leaves and indicated that this enzyme is enriched in hydrophobic residues.

TL;DR: Closure of stomata in the incompatible interaction of soybean cultivar Harosoy with race 1 with P. sojae is apparently brought about by a transmissible signal that is derived from cells that react hypersensitively to pathogen invasion.

TL;DR: Induction ofβ-1,3-glucanase activity was observed in all lines, including Palmiet, concurrent with the formation of substomatal vesicles and subsequent differentiation of the pathogen, and the hypothesis that resistance in lines carrying Lr29 or Lr34 is related to higher levels of β-1-3- glucanases expression was not confirmed.

TL;DR: The hypothesis that a lipoxygenase with chloroplastic location is induced in the incompatible interaction, and which is responsible for the increase in lipid peroxidation is advanced.

TL;DR: The results suggest the presence of a gene-for-gene interaction between F. lycopersici -tomato and F. melonis -muskmelon, in which cross protection against a virulent race is mediated by recognition of a specific elicitor from the avirulent race by the plant resistance gene product and by subsequent induction of the plant defense reaction.

TL;DR: The competitive ELISA method developed in this study, relatively abundant quantities of the toxins, i.e. approximately 0.1 mg of syringopeptins per gram fresh tissue, were detected when an aqueous extract from cotyledons of zucchini infected with a strain of P. lachrymans was tested.