Journal ArticleDOI
Cancer Mortality Among Diabetics
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This article is published in Journal of the National Cancer Institute.The article was published on 1970-03-01. It has received 239 citations till now. The article focuses on the topics: Cancer Death Rate & Standardized mortality ratio.read more
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Diabetes increases the risk of chronic liver disease and hepatocellular carcinoma
TL;DR: Among men with diabetes, the risk of chronic nonalcoholic liver disease and hepatocellular carcinoma is doubled, independent of alcoholic liver disease, viral hepatitis, or demographic features.
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Type-II diabetes and pancreatic cancer: a meta-analysis of 36 studies.
Rachel R. Huxley,Alireza Ansary-Moghaddam,A Berrington de González,Federica Barzi,M N Woodward +4 more
TL;DR: A modest causal association between type-II diabetes and pancreatic cancer is supported and is explained, in part, by higher risks being reported by smaller studies and studies that reported before 2000.
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Diabetes Mellitus and Risk of Colorectal Cancer: A Meta-Analysis
TL;DR: A meta-analysis of published data on the association between diabetes and the incidence and mortality of colorectal cancer found that diabetes was associated with an increased risk of colors, compared with no diabetes, and these results were consistent between case-control and cohort studies and between studies conducted in the United States and in Europe.
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Insulin, Insulin-Like Growth Factors and Colon Cancer: A Review of the Evidence
TL;DR: Many studies indicate that dietary patterns that stimulate insulin resistance or secretion, including high consumption of sucrose, various sources of starch, a high glycemic index and high saturated fatty acid intake, are associated with a higher risk of colon cancer.
Journal ArticleDOI
Hormonal etiology of epithelial ovarian cancer, with a hypothesis concerning the role of androgens and progesterone.
TL;DR: The purpose of this review is to evaluate evidence for and against the incessant ovulation and gonadotropin hypotheses, as well as to consider the possibility that risk of ovarian cancer may be increased by factors associated with excess androgenic stimulation of ovarian epithelial cells and may be decreased by factors related to greater progesterone stimulation.