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Open AccessJournal ArticleDOI

Efficacy of single anastomosis sleeve ileal (SASI) bypass for type-2 diabetic morbid obese patients: Gastric bipartition, a novel metabolic surgery procedure: A retrospective cohort study.

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TLDR
SASI bypass is a promising operation that offers excellent weight loss and diabetic resolution and is a therapeutic option for obese T2DM patients.
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This article is published in International Journal of Surgery.The article was published on 2016-10-01 and is currently open access. It has received 71 citations till now. The article focuses on the topics: Sleeve gastrectomy & Anastomosis.

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IFSO Worldwide Survey 2016: Primary, Endoluminal, and Revisional Procedures.

TL;DR: There was an increase in the total number both of surgical and endoluminal bariatric/metabolic procedures performed worldwide in 2016 and the surgical trends from 2008 to 2016.
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Standardization of Bariatric Metabolic Procedures: World Consensus Meeting Statement

TL;DR: Standard versions of the finished anatomic configurations of 22 surgical procedures were established by expert consensus, as a first step in developing evidence-based standard bariatric metabolic surgical procedures with the aim of improving consistency in surgery, data collection, comparison of procedures, and outcome reporting.
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Evaluation of the Efficacy of Single Anastomosis Sleeve Ileal (SASI) Bypass for Patients with Morbid Obesity: a Multicenter Study.

TL;DR: The SASI bypass is an effective bariatric and metabolic surgery that achieved satisfactory weight loss and improvement in medical comorbidities, including T2DM, hypertension, sleep apnea, and GERD, with a low complication rate.
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Single anastomosis sleeve ileal (SASI) bypass versus sleeve gastrectomy: a case-matched multicenter study

TL;DR: Both procedures had similar weight loss at 6 months postoperatively and comparable complication rates and SASI bypass conferred better improvement in T2DM and GERD than SG.
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1-Year Follow-up of Single Anastomosis Sleeve Ileal (SASI) Bypass in Morbid Obese Patients: Efficacy and Concerns

TL;DR: SASI bypass is a newly introduced investigational procedure for improving weight loss and comorbidities; however, it may result in EWL and protein malnutrition and should only be performed for select patients and by well-experienced bariatric surgeons.
References
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Journal ArticleDOI

High STOP-Bang score indicates a high probability of obstructive sleep apnoea

TL;DR: In the surgical population, a STOP-Bang score of 5–8 identified patients with high probability of moderate/severe OSA and can help the healthcare team to stratify patients for unrecognized OSA, practice perioperative precautions, or triage patients for diagnosis and treatment.
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Neuroendocrine and peripheral activities of ghrelin: implications in metabolism and obesity.

TL;DR: The theoretical possibility that GHS analogues could become candidate drugs for treatment of pathophysiological conditions in internal medicine totally unrelated to disorders of GH secretion is confronted, particularly, GHS receptor agonists or antagonists acting on appetite could represent new drug intervention in eating disorders.
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Attenuated GLP-1 secretion in obesity: cause or consequence?

TL;DR: Postprandial GLP-1 secretion in response to oral carbohydrate is considerably attenuated in obese subjects and it is suggested that both this fall and the overall reduction in GLp-1 values in obese Subjects may be related to an increase in plasma non-esterified fatty acids.
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Laparoscopic Roux-en-Y gastric bypass: 10-year follow-up.

TL;DR: Ten-year follow-up results with laparoscopic Roux-en-Y gastric bypass with hand-sewn gastrojejunal anastomosis in a group private practice show sustainable weight loss and resolution of co-morbidities, however, nutritional deficiencies presented sporadically over time and underscore the importance of routine testing.
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The gut hormone peptide YY regulates appetite.

TL;DR: It was found that peripheral administration of PYY3–36 inhibited food intake in rodents and increased C‐Fos immunoreactivity in the arcuate nucleus, suggesting that PYY 3–36 may signal food ingestion from the gut to appetite‐regulating circuits within the brain.
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