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How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease
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The article was published on 2010-01-01 and is currently open access. It has received 1442 citations till now. The article focuses on the topics: Tobacco smoke.read more
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Journal ArticleDOI
Feasibility of identifying the tobacco-related global metabolome in blood by UPLC-QTOF-MS.
Ping-Ching Hsu,Bin Zhou,Yi Zhao,Habtom W. Ressom,Amrita K. Cheema,Wallace B. Pickworth,Peter G. Shields +6 more
TL;DR: In addition to the identification of nicotine metabolites as expected, there was a characteristic profile distinguishing smokers from nonsmokers, showing the potential to identify metabolic phenotypes and new metabolites relating to cigarette smoke exposure and toxicity.
Journal ArticleDOI
Effects of MAO inhibition and a combination of minor alkaloids, β-carbolines, and acetaldehyde on nicotine self-administration in adult male rats.
Tracy T. Smith,Matthew B. Schaff,Laura E. Rupprecht,Rachel L. Schassburger,Deanne M. Buffalari,Sharon E. Murphy,Alan F. Sved,Eric C. Donny +7 more
TL;DR: The data suggest that this cocktail of constituents does not significantly alter the primary reinforcing effects of nicotine, but constituents that inhibit MAO may increase the primary reinforcement effects of Nicotine, especially at low doses.
Posted Content
Public-place Smoking Laws And Exposure To Environmental Tobacco Smoke (ets)
TL;DR: The authors found that smoking restrictions had no effect on smoking but induced large and statistically significant reductions in public-place ETS exposure, especially in bars and restaurants, and did not find significant evidence of ETS displacement to private homes.
Journal ArticleDOI
Anxiety disorders and onset of cardiovascular disease: the differential impact of panic, phobias and worry.
TL;DR: Baseline generalized anxiety disorder was strongly associated with onset of CVD (adjusted OR: 3.39).
Journal ArticleDOI
Untargeted metabolomic profiling in saliva of smokers and nonsmokers by a validated GC-TOF-MS method.
TL;DR: Most of the identified metabolites, including tyramine, adenosine, and glucose-6-phosphate, could be linked to smoking-related perturbations and may be associated with established detrimental effects of smoking.