A
Aamir Ahmad
Researcher at University of Alabama at Birmingham
Publications - 257
Citations - 15418
Aamir Ahmad is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Cancer & Cancer stem cell. The author has an hindex of 63, co-authored 251 publications receiving 13404 citations. Previous affiliations of Aamir Ahmad include Aligarh Muslim University & Hamad Medical Corporation.
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Journal ArticleDOI
Coinage Metal Complexes Against Breast Cancer
TL;DR: This review takes stock of the latest developments in the field of coinage metal anticancer drugs with an emphasis on their biological and mechanistic aspects.
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3,3'-Diindolylmethane enhances taxotere-induced apoptosis in hormone-refractory prostate cancer cells through survivin down-regulation.
TL;DR: Inactivation of survivin by DIM enhanced the therapeutic efficacy of Taxotere in prostate cancer in general, which could be useful for the treatment of HRPC and metastatic prostate cancer.
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Plumbagin induces cell death through a copper-redox cycle mechanism in human cancer cells
S. Nazeem,Asfar S. Azmi,Sarmad Hanif,Aamir Ahmad,Ramzi M. Mohammad,S.M. Hadi,K. Sateesh Kumar +6 more
TL;DR: This study is the first to investigate the copper-mediated anticancer mechanism of plumbagin in human cancer cells and these properties could be further explored for the development of anticancer agents with higher therapeutic indices, especially for skin cancer.
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Erlotinib resistance in lung cancer: current progress and future perspectives.
TL;DR: Evidently, new chemotherapy strategies are desperately needed in order to better treat lung cancer patients, and current research is investigating alternative treatment plans to enhance the chemotherapy that is already offered.
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Hydroxytyrosol Induces Apoptosis and Cell Cycle Arrest and Suppresses Multiple Oncogenic Signaling Pathways in Prostate Cancer Cells.
Haseeb Zubair,Arun Bhardwaj,Aamir Ahmad,Sanjeev K. Srivastava,Mohammad Aslam Khan,Girijesh Kumar Patel,Seema Singh,Ajay P. Singh +7 more
TL;DR: The results establish a pleiotropic activity of HT against these oncogenic signaling pathways against Castration-resistant prostate cancers, which support further testing of HT for prostate cancer therapy.