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Anders Sönnerborg

Researcher at Karolinska Institutet

Publications -  432
Citations -  21283

Anders Sönnerborg is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Viral load & Virus. The author has an hindex of 66, co-authored 407 publications receiving 19240 citations. Previous affiliations of Anders Sönnerborg include University of Missouri & University College London.

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Journal ArticleDOI

Immunological changes in primary HIV-1 infection.

Hans Gaines, +9 more
- 01 Oct 1990 - 
TL;DR: It is suggested that the infection, following the acute symptomatic stage, usually enters a stage of chronic active rather than latent infection.
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Full-length sequence of an ethiopian human immunodeficiency virus type 1 (HIV-1) isolate of genetic subtype C.

Mika Salminen, +7 more
- 20 Sep 1996 - 
TL;DR: Clone C2220, 9031 nt in length, was derived by long PCR amplification of proviral DNA from virus cultured on primary peripheral blood mononuclear cells, and contains all but 74 nt of the unique sequence information of the HIV-1 genome.
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Severe functional impairment and elevated PD‐1 expression in CD1d‐restricted NKT cells retained during chronic HIV‐1 infection

Markus Moll, +7 more
- 01 Mar 2009 - 
TL;DR: The data demonstrate a severe functional impairment in the remaining NKT‐cell compartment in HIV‐1‐infected patients, which limits the prospects to mobilize these cells in immunotherapy approaches in patients.
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Long Terminal Repeat Promoter/Enhancer Activity of Different Subtypes of HIV Type 1

Mojgan H. Naghavi, +4 more
- 20 Sep 1999 - 
TL;DR: The data suggest that genetic diversity of the LTR may result in HIV-1 subtypes with different replicative properties, and several subtype-specific LTR sequences of the various HIV- 1 strains were found.
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Effect of N-acetylcysteine(NAC) treatment on HIV-1 infection: a double-blind placebo-controlled trial.

B. Åkerlund, +5 more
- 01 Jan 1996 - 
TL;DR: NAC had no effect on the radical production by neutrophils, and although it did not increase the CD4+ cell count, it may have decreased the decline inCD4+ cells.