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Andy R Ness

Researcher at University Hospitals Bristol NHS Foundation Trust

Publications -  425
Citations -  38919

Andy R Ness is an academic researcher from University Hospitals Bristol NHS Foundation Trust. The author has contributed to research in topics: Body mass index & Population. The author has an hindex of 87, co-authored 415 publications receiving 35275 citations. Previous affiliations of Andy R Ness include National Institute for Health Research & University of Bristol.

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Supplementation with a low-moderate dose of n-3 long-chain PUFA has no short-term effect on bone resorption in human adults.

TL;DR: The findings provide no evidence for an association between n-3 PUFA supplementation (1·48 g EPA+DHA/d) for 12 weeks and bone resorption in humans assessed by β-CTX, and suggest that n-2 PUFA supplements may be unlikely to be of benefit in preventing bone loss.
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Peripubertal changes in circulating antimüllerian hormone levels in girls.

TL;DR: Measurement of circulating AMH and inhibin B levels suggests that the rate of ovarian primordial follicle recruitment increases in the prepubertal years then declines again following the onset of puberty as follicular activity pattern changes.
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Common Variation in the WNK1 Gene and Blood Pressure in Childhood: The Avon Longitudinal Study of Parents and Children

TL;DR: The findings show that common intronic and exonic WNK1 variants are associated with diastolic blood pressure gradient from 7 to 11 years and with systolicBlood pressure at 11 years.
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The Role of Diet, Fruit and Vegetables and Antioxidants in the Aetiology of Stroke:

TL;DR: Diet provides the most plausible candidate to explain these international and temporal variations in deaths attributed to stroke.
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Maternal anemia, iron intake in pregnancy, and offspring blood pressure in the Avon Longitudinal Study of Parents and Children

TL;DR: Some evidence indicates that maternal anemia in contemporary pregnant women is associated with lower offspring BP, and it is possible that, in well-nourished populations, low hemoglobin is more likely to reflect greater plasma volume expansion (and thus better maternal and offspring health) than iron deficiency.