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Anthony S.L. Tang
Researcher at University of British Columbia
Publications - 145
Citations - 11655
Anthony S.L. Tang is an academic researcher from University of British Columbia. The author has contributed to research in topics: Heart failure & Cardiac resynchronization therapy. The author has an hindex of 44, co-authored 135 publications receiving 10830 citations. Previous affiliations of Anthony S.L. Tang include University of Ottawa & Duke University.
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Journal ArticleDOI
Cardiac-Resynchronization Therapy for Mild-to-Moderate Heart Failure
Anthony S.L. Tang,George A. Wells,Mario Talajic,Malcolm Arnold,Robert S. Sheldon,Stuart J. Connolly,Stefan H. Hohnloser,Graham Nichol,David H. Birnie,John L. Sapp,Raymond Yee,Jeff S. Healey,Jean L. Rouleau +12 more
TL;DR: Among patients with NYHA class II or III heart failure, a wide QRS complex, and left ventricular systolic dysfunction, the addition of CRT to an ICD reduced rates of death and hospitalization for heart failure.
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Effects of physiologic pacing versus ventricular pacing on the risk of stroke and death due to cardiovascular causes
Stuart J. Connolly,Charles R. Kerr,Michael Gent,Robin S. Roberts,Salim Yusuf,Anne M. Gillis,Magdi Sami,Mario Talajic,Anthony S.L. Tang,George Klein,Ching Lau,David Newman +11 more
TL;DR: Physiologic pacing provides little benefit over ventricular pacing for the prevention of stroke or death due to cardiovascular causes and the effect on the rate of atrial fibrillation was not apparent until two years after implants.
Journal ArticleDOI
Identification of a Gene Responsible for Familial Wolff–Parkinson–White Syndrome
Michael H. Gollob,Martin S. Green,Anthony S.L. Tang,Gollob T,Akihiko Karibe,Ali Hassan As,Ferhaan Ahmad,Lozado R,Shah G,Fananapazir L,Linda L. Bachinski,Robin S. Roberts +11 more
TL;DR: Two families in which the Wolff–Parkinson–White syndrome segregated as an autosomal dominant disorder are identified, and candidate genes were identified, sequenced, and analyzed in normal and affected family members to identify the disease-causing gene.
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Ventricular Tachycardia Ablation versus Escalation of Antiarrhythmic Drugs
John L. Sapp,George A. Wells,Ratika Parkash,William G. Stevenson,Louis Blier,Jean-François Sarrazin,Bernard Thibault,Lena Rivard,Lorne J. Gula,Peter Leong-Sit,Vidal Essebag,Pablo B. Nery,Stanley Tung,Jean-Marc Raymond,Laurence D. Sterns,George D. Veenhuyzen,Jeff S. Healey,Damian P. Redfearn,Jean-Francois Roux,Anthony S.L. Tang +19 more
TL;DR: There was a significantly lower rate of the composite primary outcome of death, ventricular tachycardia storm, or appropriate ICD shock among patients undergoing catheter ablation than among those receiving an escalation in antiarrhythmic drug therapy.
Journal ArticleDOI
Electrophysiologic testing to identify patients with coronary artery disease who are at risk for sudden death
Alfred E. Buxton,Kerry L. Lee,Lorenzo A. DiCarlo,Michael R. Gold,G S Greer,Eric N. Prystowsky,Michael F. O'Toole,Anthony S.L. Tang,John D. Fisher,James Coromilas,Mario Talajic,Gail E. Hafley +11 more
TL;DR: Electrophysiologic testing in patients who had coronary artery disease, a left ventricular ejection fraction of 40 percent or less, and asymptomatic, unsustained ventricular tachycardia found them to have a significantly lower risk of sudden death or cardiac arrest and lower overall mortality than similar patients with inducible sustained tachyarrhythmias.