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Barbara Casadei
Researcher at University of Oxford
Publications - 274
Citations - 23925
Barbara Casadei is an academic researcher from University of Oxford. The author has contributed to research in topics: Atrial fibrillation & Medicine. The author has an hindex of 60, co-authored 242 publications receiving 19620 citations. Previous affiliations of Barbara Casadei include Newman University & King's College London.
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Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
TL;DR: A key role for nNOS is shown in the control of basal and adrenergically stimulated cardiac contractility and in the autonomic control of heart rate, whether or not the role of nN OS carries implications for cardiovascular disease remains an intriguing possibility requiring future study.
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European Society of Cardiology smartphone and tablet applications for patients with atrial fibrillation and their health care providers
Dipak Kotecha,Winnie Chua,Larissa Fabritz,Jeroen M.L. Hendriks,Jeroen M.L. Hendriks,Barbara Casadei,Ulrich Schotten,Panos E. Vardas,Hein Heidbuchel,Veronica Dean,Paulus Kirchhof +10 more
TL;DR: The objective is to demonstrate the value of integrating novel digital technology into clinical practice, with the potential for patient engagement, optimization of pharmacological and interventional therapy in AF, and ultimately to improve patient outcomes.
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Nitric-oxide-mediated regulation of cardiac contractility and stretch responses.
Barbara Casadei,Claire E. Sears +1 more
TL;DR: The role of myocardial NO production in regulating cardiac function appears to be more complex and controversial as mentioned in this paper, and some studies have shown that non-isoform-specific inhibition of NO synthesis with l -arginine analogues has no effect on basal contraction in LV myocytes.
Journal Article
Nitric oxide-mediated regulation of cardiac contractility and stretch responses
TL;DR: The findings suggest that NO can mediate diverse and even contrasting actions within the myocardium, a notion that is difficult to reconcile with the early description of NO as a highly reactive and diffusible molecule possessing minimal specificity in its interactions.
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Targeting Inflammation and Oxidative Stress in Atrial Fibrillation: Role of 3-Hydroxy-3-Methylglutaryl-Coenzyme A Reductase Inhibition with Statins
TL;DR: Evidence indicates that statins prevent AF-induced electrical remodeling in animal models of atrial tachypacing and may reduce the new onset of AF after cardiac surgery.