B
Bert Binas
Researcher at Hanyang University
Publications - 55
Citations - 1563
Bert Binas is an academic researcher from Hanyang University. The author has contributed to research in topics: Embryonic stem cell & Fatty acid-binding protein. The author has an hindex of 18, co-authored 49 publications receiving 1441 citations. Previous affiliations of Bert Binas include Texas A&M University & Max Delbrück Center for Molecular Medicine.
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Journal ArticleDOI
Preimplantation-stage stem cells induce long-term allogeneic graft acceptance without supplementary host conditioning
Fred Fändrich,X. Lin,Gui X. Chai,Gui X. Chai,Maren Schulze,Detlev Ganten,Michael Bader,Julia U Holle,D.S Huang,Reza Parwaresch,Nicholaus Zavazava,Bert Binas,Bert Binas +12 more
TL;DR: It is shown that when rat embryonic stem cell-like cells of WKY origin are injected intraportally into fully MHC-mismatched DA rats, they engraft permanently without supplementary host conditioning, which sets the basis for long-term graft acceptance of second-set transplanted WKY cardiac allografts.
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Decreased liver fatty acid binding capacity and altered liver lipid distribution in mice lacking the liver fatty acid-binding protein gene.
Gregory G. Martin,Heike Danneberg,Leena S. Kumar,Barbara P. Atshaves,Erdal Erol,Michael Bader,Friedhelm Schroeder,Bert Binas +7 more
TL;DR: A quantitative assessment of the contribution of L-FABP to cytosolic fatty acid binding capacity is provided and it is suggested that SCP-2 contributes to the accumulation of cholesterol in L- FABP null liver.
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Liver fatty acid-binding protein initiates budding of pre-chylomicron transport vesicles from intestinal endoplasmic reticulum.
Indira Neeli,Shadab A. Siddiqi,Shahzad Siddiqi,James Mahan,William S. Lagakos,Bert Binas,Tarun Gheyi,Judith Storch,Charles M. Mansbach,Charles M. Mansbach +9 more
TL;DR: It is concluded that L-FABP can select cargo for and bud PCTV from intestinal ER membranes in the absence of the requirement for COPII proteins.
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Liver fatty acid-binding protein is required for high rates of hepatic fatty acid oxidation but not for the action of PPAR-α in fasting mice
TL;DR: The results suggest that under fasting conditions, hepatic L‐FABP contributes to hepatic LCFA oxidation and ketogenesis by a nontranscriptional mechanism, whereas L‐fABP can activate ketogenic gene expression in fed mice, whereas the mechanisms whereby L‐ FABP affects fatty acid oxidation may vary with physiological condition.
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Heart-Type Fatty Acid Binding Protein Regulates Dopamine D2 Receptor Function in Mouse Brain
TL;DR: It is shown that H-FABP is highly expressed in acetylcholinergic interneurons and terminals of glutamatergic neurons in the dorsal striatum of mouse brain but absent in dopamine neuron terminals and spines in the same region, thereby regulating dopamine D2R function in the striatum.