scispace - formally typeset
B

Brooke S. G. Molina

Researcher at University of Pittsburgh

Publications -  84
Citations -  9413

Brooke S. G. Molina is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Attention deficit hyperactivity disorder & Poison control. The author has an hindex of 38, co-authored 74 publications receiving 8374 citations. Previous affiliations of Brooke S. G. Molina include University of Washington & Arizona State University.

Papers
More filters
Journal ArticleDOI

The MTA at 8 Years: Prospective Follow-up of Children Treated for Combined-Type ADHD in a Multisite Study

TL;DR: Type or intensity of 14 months of treatment for ADHD in childhood does not predict functioning 6 to 8 years later, and early ADHD symptom trajectory regardless of treatment type is prognostic, implying that children with behavioral and sociodemographic advantage, with the best response to any treatment, will have the best long-term prognosis.
Journal ArticleDOI

Childhood predictors of adolescent substance use in a longitudinal study of children with ADHD.

TL;DR: Children diagnosed with attention-deficit/hyperactivity disorder were prospectively monitored into adolescence to evaluate their risk for elevated substance use relative to same-aged adolescents without ADHD, and persistence of ADHD and adolescent CD were each associated with elevated substanceUse behaviors relative to controls.
Journal ArticleDOI

Attention-deficit hyperactivity disorder and problems in peer relations: predictions from childhood to adolescence.

TL;DR: Impairments in peer relations for ADHD youths, known to be common in childhood, also exist in adolescence and further research into the causes and treatment of poor social functioning in youths with ADHD is recommended.
Journal ArticleDOI

Relation of parental alcoholism to early adolescent substance use: A test of three mediating mechanisms.

TL;DR: Results suggested that parental alcoholism influenced adolescent substance use through stress and negative affect pathways, through decreased parental monitoring, and through increased temperamental emotionality (which was associated with heightened negative affect).