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Bruce L. Miller

Researcher at University of California, San Francisco

Publications -  1296
Citations -  135366

Bruce L. Miller is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Frontotemporal dementia & Dementia. The author has an hindex of 163, co-authored 1153 publications receiving 115975 citations. Previous affiliations of Bruce L. Miller include University of Southern California & National Institutes of Health.

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Of brain and bone: The unusual case of Dr. A

TL;DR: This case demonstrates that social and emotional dysfunction in FTD can be dissociated from preserved performance on classic executive functioning tasks and suggests a possible genetic link between bone disease and FTD.
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Concordance and discordance between brain perfusion and atrophy in frontotemporal dementia

TL;DR: The results suggest that damage of brain function in FTD, assessed by ASL perfusion, can vary regionally despite widespread atrophy, and detection of discordance between brain perfusion and structure inFTD might aid diagnosis and staging of the disease.
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Language and spatial dysfunction in Alzheimer disease with white matter thorn-shaped astrocytes.

TL;DR: The negative impact of WM-TSA pathology to language and possibly visuospatial networks suggests that WM- TSA is not as benign as other ARTAG types and may be explored as a framework to understand the mechanisms and impact of astrocytic tau deposition in AD in humans.
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Characterizing radiology reports in patients with frontotemporal dementia

TL;DR: A retrospective analysis of MRI atrophy patterns in 40 patients with bvFTD found nine categories were identified based on the radiologist’s reports of the 40 patients: 1) bv FTD, 2) white matter/ischemic disease, 3) AD, 4) normal pressure hydrocephalus/hydrocephalus, 5) mitochondrial/metabolic, 6) encephalomalacia, …
Journal Article

Psychometric and SPECT studies in alzheimer’s disease with and without delusions

TL;DR: In the setting of AD, right temporal function deterioration may contribute to the development of delusions, and patients who developed delusions showed deterioration in right anterior temporal rCBF compared to AD without delusions.